How does vomiting lead to low urine Cl-. Isn't because low volume triggers angiotensin II, angiotensin II increases sodium reabsorption, which thereby increases chloride reabsorption in the late proximal tubule?
Would ang II also stimulatie bicarb reabsorption in the early PT too? Wouldn't this make the alkalosis worse?
Would ang II also stimulatie bicarb reabsorption in the early PT too? Wouldn't this make the alkalosis worse?