One of aldosterone's primary effects is the resorption of Na+ from the kidney's. Na+ is reabsorbed and then water follows. Is the solution is resorbed from the kidneys to the ECF and isotonic or hypertonic solution?
I wish I could put to words my frustration that my long, drawn out post didn't show up when I posted it yesterday.
Here are the cliff notes:
Aldosterone increases Na+ reabsorption and K+ secretion. The hypernatremia is not a result of a change in plasma tonicity. What actually happens is that the Na+ reabsorption causes osmolality receptors to send signals to release ADH(the real hormone in charge of osmoregulation) and this allows water to follow the reabsorbed Na+ passively out of the collecting duct in to the plasma.
Thank you all for the well-written help, especially Physics. I have a very good understanding of it now, but my original question is a little bit murky still.
A lot of aldosterone would give you hyperaldosteronism so yes.True of false: aldosteronism results in hyperaldosteronism.
true.True or false: aldosterone only resorbs Na+, no water follows via osmosis
For your original question: the final result is that when the aldosterone is released the fluid reabsorbed from the kidney is isotonic to blood.
BRS physio didn't change much between editions. Don't worry about it. And don't focus too much on the finer details of Neurophysiology either, that section is overkill for the boards.since this is a physio topic I have a question.
I have BRS physio second edition..is the third edition better? maybe more graphics?