Don't fret! It's probably beyond the scope of the test, but Gitelman syndrome is pretty much the same as a thiazide diuretic. Whereas the diuretic blocks the channel, Gitelman is a defect in the channel itself. So same effects.
Really sorry. Could you clarify the actual nephron physiology? Like maybe flow chart of channels or something. I'm looking at it and something is probably escaping my mind. The way I understood it via Kaplan videos and the explanation of thiazide diuretics is that if the Na+/Cl- channel is open, sodium is being reabsorbed which will, along with the Na+/K+ ATPase pump on the basolateral side, create a gradient for the Na+/Ca2+ exchanger and increase levels of Ca2+ in the blood. But blocking it is actually what increases calcium?