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How long does aderoll stay in system?

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Conquistador

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Sounds like alot of people are taking aderoll. It might be helpful to start a thread on the negatives of the drug. How long after use is it detectable on tests? What are common side effects? What are interaction warnings i.e, alcohol, caffeine, crack(hehe), etc. We've heard the positives, what are the dangers?
 

Doctor Octopus

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SIDE EFFECTS

Cardiovascular: Palpitations, tachycardia, elevation of blood pressure There have been isolated reports of cardiomyopathy associated with chronic amphetamine use.

Central Nervous System: Psychotic episodes at recommended doses (rare), overstimulation, restlessness. dizziness, insomnia, euphoria. dyskinesia, dysphoria, tremor, headache, exacerbation of motor and phonictics and Tourette's syndrome.

Gastrointestinal: Dryness of the mouth, unpleasant taste, diarrhea, constipation, other gastrointestinal disturbances. Anorexia and weight loss may occur as undesirable effects when amphetamines are used for other than the anorectic effect.

Allergic: Urticaria.

Endocrine: Impotence. Changes in libido.


DRUG INTERACTIONS

Acidifying agents - Gastrointestinal acidifying agents (guanethidine,reserpine, glutamic acid HCl,ascorbic acid, fruit juices, etc.) lower absorption of amphetamines.

Urinary acidifying agents -(ammonium chloride, sodium acid phosphate, etc.) Increase the concentration of the ionized species of the amphetamine.

Primary excretion - Both Groups of agents lower blood levels and efficacy of amphetamines.

Adrenergic blockers - Adrenergic blockers are inhibited by amphetamines.

Alkalinizing agents -Gastrointestinal alkalinizing agents (sodium bicarbonate, etc.)increase absorption of amphetamines. Urinary alkalinizing agents (acetazolamide, some thiazides) increase the concentration of the non-ionized species of the amphetamine molecule, thereby decreasing urinary excretion. Both groups of agents increase blood levels and therefore potentate the actions of amphetamines.

Antidepressants, tricyclic - Amphetamines may enhance the activity of tricyclic or sympathomimetic agents; d-amphetamine with desipramine or protriptyline and possibly other tricyclics cause striking and sustained increases in the concentration of d-amphetamine in the brain; cardiovascular effects can be potentiated.

MAO inhibitors - MAO antidepressants, as well as a metabolite of furazolidone, slow amphetamine metabolism. This slowing potentiates amphetamines, increasing their effect on the release of norepinephrine and other monoamines from adrenergic nerve endings, this can cause headaches and other signs of hypertensive crisis. A variety of neurological toxic effects and malignant hyperpyrexia can occur, sometimes with fatal results.

Antihistamines - Amphetamines may counteract the sedative effect of antihistamines.

Antihypertensives - Amphetamines may antagonize the hypotensive effects of antihypertensives.

Chlorpromazine - Chlorpromazine blocks dopamine and norepinephrine receptors, thus inhibiting the central stimulant effects of amphetamines, and can be used to treat amphetamine poisoning.

Ethosuximide - Amphetamines may delay intestinal absorption of ethosuximide.

Haloperidol - Haloperidol blocks dopamine receptors, thus inhibiting the central stimulant effects of amphetamines.

Lithium carbonate - The anorectic and stimulatory effects of amphetamines may be inhibited by lithium carbonate.

Meperidine - Amphetamines pone the analgesic effect of meperidine.

Methenamine therapy - Urinary excretion of amphetamines is increased, and efficacy is reduced, by acidifying agents used in methenamine therapy.

Norepinephrine - Amphetamines enhance the adrenergic effect of norepinephrine.

Phenobarbital - Amphetamines may delay intestinal absorption of phenobarbital; co-administration of phenobarbital may produce a synergistic anticonvulsant action.

Phenytoin - Amphetamines may delay intestinal absorption of phenytoin; co-administration of phenytoin may produce a synergistic anticonvulsant action.

Propoxyphene - In cases of propoxyphene overdose, amphetamine CNS stimulation is potentiated and fatal convulsions can occur.

Veratrum alkaloids - Amphetamines inhibit the hypotensive effect of veratrum alkaloids.

Drug/Laboratory Test Interactions

Amphetamines can cause a significant elevation in plasma corticosteroid levels. This increase is greatest in the evening.

Amphetamines may interfere with urinary steroid determinations
 

Doctor Octopus

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Amphetamines Have A High Potential For Abuse. Administration Of Amphetamines For Prolonged Periods Of Time May Lead To Drug Dependence And Must Be Avoided. Particular Attention Should Be Paid To The Possibility Of Subjects Obtaining Amphetamines For Non Therapeutic Use Or Distribution To Others, And The Drugs Should Be Prescribed Or Dispensed Sparingly.
 

Doctor Octopus

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Sure. I think as far as staying in your system the half-life is 8-10 hours and will be undectebable in the urine after 72 hours. Another things is that the psychotic episodes, while rare at therapeutic doses, become common (almost guaranteed) at much higher doses. It's good to know this ahead of time if you're going to get high with this stuff (bad idea).
 
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