Amrita, Drmon is right.
The portion of the nucleus that innervates the muscles of the forehead receives corticobulbar fibers from both the contralateral and ipsilateral motor cortex.
The portion of the nucleus that innervates the lower muscles of facial expression receives corticobulbar fibers from only the contralateral motor cortex.
This is very important clinically as central (upper motor neuron) and peripheral (lower motor neuron) lesions will present differently.
With an UMN lesion, voluntary control of only the lower muscles of facial expression on the side contralateral to the lesion will be lost.
Voluntary control of muscles of the forehead will be spared due to the bilateral innervation of the portion of the motor nucleus of CN VII that innervates the upper muscles of facial expression.
A LMN lesion results in the paralysis of all muscles of facial expression (including those of the forehead) ipsilateral to the lesion.
A LMN lesion of CN VII which occurs at or beyond the stylomastoid foramen is commonly referred to as a Bell's Palsy.Characteristic indications of a LMN lesion or Bell's Palsy on the affected side.
In UMN, spastic paralysis - hyperreflexia & disuse atrophy of skeletal muscle.
In LMN, flaccid paralysis - Areflexia,fasciculations & atrophy.