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- Jun 25, 2013
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Is it the valsalva maneuver straining phase or the relaxing phase that increases cardiac parasympathetic tone?
what i know:
straining phase - decreases venous return/ventricular volume, increased thoracic/ central venous pressure
release phase - increases venous return/ ventricular volume, decreased thoracic/central venous pressure
confusion:
logic would say that it's the release phase that increases pressure because of increased volume flowing through/stretching baroreceoptors
then that means when we use valsalva to help with paroxymal supraventricular tachycardia, we are really getting the decreased vagal tone benefit from the valsalva release phase and NOT the straining phase. and somewhat the neural magic (solitary nucleus) of the release phase overpowers the previous neural magic of the straining phase (with the decreased VR business).
Right?
what i know:
straining phase - decreases venous return/ventricular volume, increased thoracic/ central venous pressure
release phase - increases venous return/ ventricular volume, decreased thoracic/central venous pressure
confusion:
logic would say that it's the release phase that increases pressure because of increased volume flowing through/stretching baroreceoptors
then that means when we use valsalva to help with paroxymal supraventricular tachycardia, we are really getting the decreased vagal tone benefit from the valsalva release phase and NOT the straining phase. and somewhat the neural magic (solitary nucleus) of the release phase overpowers the previous neural magic of the straining phase (with the decreased VR business).
Right?