Why is tachypnea a sign of fluid overload?

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my thoughts would be Pulm. edema would involve pressure changes favoring collapse of alveoli/atelectasis. With the loss of functional alveoli you'd lose surface area for gas exchange (ain't breathin' so good), and therefore increasing PaCO2 and decreasing PaO2, the former would stimulate central chemoreceptors in the medulla of the brain stem (respiratory groups) to increase rate of breathing to correct the blood gases. So it would be a compensatory tachypnea.
 
Can someone please explain the pathophysiology behind this.

Thanks in advance.

multiple reasons:
1) With left sided volume overload, you get pulmonary edema which causes increased dead space, V/Q mismatch, hypoxia --> tachypnea

2) You see worsening of diffusion, DLCO worsens such that any movement causes worsening desaturation more than the V/Q mismatch alone.
 
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