Now this is a very confusing topic for me
The genesis of ST elevation elevation and hyperacute T waves can be explained on the basis of the systolic and diastolic current theory
But how do you explain Tinversion ans ST depression.?
In the small subset of subendocardial ischemia, the ischemic subendocardial cells remain less depolarised than the normal epicardial cells, so the vector will be directed away from the epicardium (ST depression in ECG).....thats all right.....But we know transmural ischemia also causes ST depression and T wave inversion-how is that????
Also in Myocardial infarcction after the initial after the initial hyperacute T, there is T inversion.....how do we explain that?????
The genesis of ST elevation elevation and hyperacute T waves can be explained on the basis of the systolic and diastolic current theory
But how do you explain Tinversion ans ST depression.?
In the small subset of subendocardial ischemia, the ischemic subendocardial cells remain less depolarised than the normal epicardial cells, so the vector will be directed away from the epicardium (ST depression in ECG).....thats all right.....But we know transmural ischemia also causes ST depression and T wave inversion-how is that????
Also in Myocardial infarcction after the initial after the initial hyperacute T, there is T inversion.....how do we explain that?????
