path quest

  • Thread starter Thread starter scsux
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scsux

in hypersensitivity type I rxn, sensitization occurs when
histamine is released from mast cells
IgE binds to mast cells
IgG binds to mast cells

Splenic embolus from
LV ??
RV
Pulmonary veins
Leg veins

Pulpal infection caused by
Candida
Treponema
Strep
Staph

Gene transfer from bacteria
Naked DNA
Plasmid
Repression???
Transduction
Bacteriophage
 
in hypersensitivity type I rxn, sensitization occurs when
histamine is released from mast cells
IgE binds to mast cells 👍
IgG binds to mast cells

Splenic embolus from
LV ??👍 not sure
RV
Pulmonary veins
Leg veins

Pulpal infection caused by
Candida
Treponema
Strep
Staph👍 not sure
plz corect if wrong
 
For staphy. Aureus are a normal habitant of Nasopharynx, but still Streptoc. is main couse of pulpal infection IMO.

Dose any one know what is common between type I hypersensitivity and type IV... the only thing that I could think of, is that both of them are result from interaction to allergic atigen, and that they are both seen in Latex hypersensitivity. But what else????
 
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