physio biochem some qs...

[awesomedds]

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84/00
cusshing syndrome is ch by which one ... inc protein anabolism / hyperkalemia(its like na re na na 50/ 50...)


85/slight detachment of retina due to decreased length from optical center lead to far sightedness or near sightedness


02
11. higest viscosity of blood in body is in vasa recta(yup.. i think so..) or vena cave????
velocity chart
aorta capillary veins
high lowest high.. am i correct....


15. what inc the rate of diffusion of O2 in alveolar membrane inc the surface area(acc to me) or dec the concentration
based on the formulla d= SArea x pressure/ thickness.... so ????


16.use of restriction endonuclease in gene cloning is when.... insertion of cDNA into vector DNA or expression of cloned gene by bacteria??? no idea....


17. role of Ca in activation of cardiac muscle... couple myosin and actin to form actinomyosin / reduce inhibition of the tropomyosin uptake in myofillaments????
can some 1 plz explain this concept of actin and myosin interaction.. what causes dossociation and whatever... or suggest a good site where i can learn and get my concept clear....

42. characteristic of creatinine are all except...freely filterd /slightly secreted /slightely reabsorbed
creatine is FF yes.. reab and secreted.. no!!!! this is not true
http://en.wikipedia.org/wiki/Creatinine_clearance


45. reduced serum K cause... hyperpolarises cell memb potential/ hypopolarised cell memb potential???? what is this..!!!!!!


59. what is essential for a sub to be amphipathic... polar -ve charged/ nonpolar + ve charged/ cyclic hydrophilic / branche hydrophilic(nut an amphipathic mol is the one which has both the philic and phobic parts.????)


58. substance responsible for initiating cross bridges in smooth muscle myosin ATPase / myosin kinase ???? again the same topic i cant get.... i feel i turned dumb!!!!!!


62. natural metabolite used to measure GFR.. inulin / creatinine
89.reverse transcriptase used to make cDNA from mRNa.. true of false... ??? if worng what do think....

plz do help.. havin a tough time studynd this hardest subject.... i wana do good so i dont have to study this terror ever again..😡

 

[drvijigobs]

84/00
cusshing syndrome is ch by which one ... inc protein anabolism / hyperkalemia(ans)(its like na re na na 50/ 50...)


85/slight detachment of retina due to decreased length from optical center lead to far sightedness or near sightedness(ans)


02
11. higest viscosity of blood in body is in vasa recta(yup.. i think so..) or vena cave????
velocity chart
aorta capillary veins
high lowest high.. am i correct....


15. what inc the rate of diffusion of O2 in alveolar membrane inc the surface area(ans)or dec the concentration
based on the formulla d= SArea x pressure/ thickness.... so ????


16.use of restriction endonuclease in gene cloning is when.... insertion of cDNA into vector DNA (ans) or expression of cloned gene by bacteria


17. role of Ca in activation of cardiac muscle... couple myosin and actin to form actinomyosin(ans) / reduce inhibition of the tropomyosin uptake in myofillaments????
can some 1 plz explain this concept of actin and myosin interaction.. what causes dossociation and whatever... or suggest a good site where i can learn and get my concept clear....

42. characteristic of creatinine are all except...freely filterd /slightly secreted (ans) /slightely reabsorbed
creatine is FF yes.. reab and secreted.. no!!!! this is not true
http://en.wikipedia.org/wiki/Creatinine_clearance


45. reduced serum K cause... hyperpolarises cell memb potential/ hypopolarised cell memb potential???? what is this..!!!!!!


59. what is essential for a sub to be amphipathic... polar -ve charged/ nonpolar + ve charged/ cyclic hydrophilic / branche hydrophilic(ans)(nut an amphipathic mol is the one which has both the philic and phobic parts.????)


58. substance responsible for initiating cross bridges in smooth muscle myosin ATPase (ans)/ myosin kinase


. natural metabolite used to measure GFR.. inulin(ans) / creatinine
89.reverse transcriptase used to make cDNA from mRNa.. true (ans)of false... ??? if worng what do think....

correct me if i'm wrong

 

[awesomedds]

plz guys hepl.... some more inputs... plzzzzzzzzzz

 

[dr37]

what inc the rate of diffusion of O2 in alveolar membrane
i think the ans should b inc the surface area, refer to the fick's law.



17. role of Ca in activation of cardiac muscle... couple myosin and actin to form actinomyosin(ans) / reduce inhibition of the tropomyosin uptake in myofillaments????

when the ap reaches the sarcolemma, it travels thru the t tubules and activates the sarcoplasmic reticulum to release Ca in the interior of the muscle cell. this Ca combines with the troponin c of the thin filaments.
this binding gets some shape changes in the troponin and exposes the myosin binding site on thin filaments i.e. actin.
hence helps in myosin actin binding....

i hope i was able to clear your doubt to some extent.....





45. reduced serum K cause... hyperpolarises cell memb potential/ hypopolarised cell memb potential???? what is this..!!!!!!

i think the ans to this ques is hyperpolarise the cell memb..
reason..
at rest the cell is said to be in a polarised state.. i.e. more k in the icf and more na in the ecf..
when an ap reaches the cell it gets depolarised i.e. rapid influx of na and efflux of k...

so if k is less in the serum i.e. less in the ecf, less negativity, more positivity i.e. hyperpolarised (remember it is already polarised when the outside is positive).
plz correct if i am wrong....

 

[dr37]

what inc the rate of diffusion of O2 in alveolar membrane
i think the ans should b inc the surface area, refer to the fick's law.



17. role of Ca in activation of cardiac muscle... couple myosin and actin to form actinomyosin(ans) / reduce inhibition of the tropomyosin uptake in myofillaments????

when the ap reaches the sarcolemma, it travels thru the t tubules and activates the sarcoplasmic reticulum to release Ca in the interior of the muscle cell. this Ca combines with the troponin c of the thin filaments.
this binding gets some shape changes in the troponin and exposes the myosin binding site on thin filaments i.e. actin.
hence helps in myosin actin binding....

i hope i was able to clear your doubt to some extent.....





45. reduced serum K cause... hyperpolarises cell memb potential/ hypopolarised cell memb potential???? what is this..!!!!!!

i think the ans to this ques is hyperpolarise the cell memb..
reason..
at rest the cell is said to be in a polarised state.. i.e. more k in the icf and more na in the ecf..
when an ap reaches the cell it gets depolarised i.e. rapid influx of na and efflux of k...

so if k is less in the serum i.e. less in the ecf, less negativity, more positivity i.e. hyperpolarised (remember it is already polarised when the outside is positive).
plz correct if i am wrong....





[/quote]

 

[s m i l e 2007]

so if k is less in the serum i.e. less in the ecf, less negativity, more positivity i.e. hyperpolarised (remember it is already polarised when the outside is positive).
its called hyperpolarized so in this stage is it easy to get AP or difficult?