Renal artery stenosis and renovascular hypertension are not synonymous. A 70% stenosis does not necessarily mean RAS is the cause of hypertension (renovascular hypertension), though it may be. Unfortunately, there isn't a 100% accurate test to prove causality here. You can use plasma renin activity, captopril-induced renal scintigraphy, etc, to help determine who may benefit from angioplasty-stenting (assuming it's atherosclerotic), but you have to be familiar with the limitations of these tests (e.g., high creatinine, bilateral stenosis, etc.). There has been a lot of research on selection of candidates who would benefit from PTAS, which you should take a look at. Unless you are part of an unfortunately not-so-small proportion of a particular specialty who practices the motto "stent who you can stent" rather than "stent who you should stent". It is sad, since a lot of this kind of research has been done by the cardiology community, but some cardiologists just seem to ignore them. Ever heard of "drive-by stenting"? If you haven't, it means putting a stent in the different branches coming off the aorta (renals, celiac, superior mesenteric, inferior mesenteric, iliac arteries) once your done stenting the coronaries. Whether the patients truly need these $tent$ or not, in the minds of some in this particular specialty, is a secondary and sometimes unimportant issue.
All that said, the particular circumstances of each patient is different, and you should consult your own doctor for patient-specific medical advice.