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So I had an older lady in septic shock last night in whom I placed a left subclavian TLC. She was frail and had easily identifiable landmarks - I got back nice dark red blood (witnessed by myself and a nurse present) on my first stick without any complications; prior to threading the wire I checked for any pulsatile flow and got only a mild dribble; placed the wire, dilator, and TLC without any difficulty and secured line at about 18 cm (she was pretty small). Drew back dark red blood on all 3 ports. Post procedure CXR showed the line to be, to my eyes, in the SVC, although prob 2-4 cm short of the SVC-RA junction (but not anywhere near the aortic knob). Fluid and PRBC flowed easily through the line with no resistance, and we had Levophed running for about 30 min to 1 hour until I saw the blood gas drawn off the line, which looked arterial - oxygen pressure was 230 and oxygen saturation was 95% (pt was on 50% FiO2 (trach/vent dependent) with pulse ox sats in the mid 90s). Concerned now that the line was arterial, we checked the pt's left radial pulse, which was strong and equal to the right. We turned off the pressors temporarily and checked simultaneous gas off the subclavian line and a gas off the R radial artery. These 2 gases were essentially the same as far as pH and pCO2, but the oxygen pressure was 130 in the radial artery stick and 170 in the subclavian line (the 2 gases were drawn no more than 2 minutes apart, with no change in vent settings or hemodynamics during this time). I rechecked the CXR, which looked like the line was in SVC. We hooked up the subclavian line as if it was an arterial line but 2/2 technical difficulties were unable to get any waveform (venous or arterial). We checked for any pulsatile flow but again there was none, and were able to drip normal saline slowly into the line without any resistance or backflow.
So my question - in the face of everything telling me the line was in the vein, except for the gases (which granted is a pretty huge except), is it possible to have essentially similar venous/arterial blood gases? My theory was that this pt had failure of her electron transport chain 2/2 sepsis and was essentially unable to utilize any oxygen (mitochondrial failure, I can't quite remember the exact name for this(, thus explaining the similarities bw the gases.
Any thoughts/similar experiences? I think that I will start transducing all lines I do now without ultrasound guidance (I think it's likely unncessary for lines that you do real time US guidance, ie IJs). Although if the flow is not pulsatile or briskly flowing when you remove the syringe prior to passing the wire, are you really going to gain anything extra by transducing?
Thanks all!
So my question - in the face of everything telling me the line was in the vein, except for the gases (which granted is a pretty huge except), is it possible to have essentially similar venous/arterial blood gases? My theory was that this pt had failure of her electron transport chain 2/2 sepsis and was essentially unable to utilize any oxygen (mitochondrial failure, I can't quite remember the exact name for this(, thus explaining the similarities bw the gases.
Any thoughts/similar experiences? I think that I will start transducing all lines I do now without ultrasound guidance (I think it's likely unncessary for lines that you do real time US guidance, ie IJs). Although if the flow is not pulsatile or briskly flowing when you remove the syringe prior to passing the wire, are you really going to gain anything extra by transducing?
Thanks all!