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Did this from kaplan, sooooo do i have to actually learn this table of death, or is kaplan enough/some other source/anything but this table please.
anticonvulsant table in FA
- Thread starter aspiringmd1015
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I took Step 1 a year ago, but unless anything's changed, a good chunk of it is unnecessary. You don't need to know the mechanism of action of most of the drugs (maybe be generally familiar with ethosuxamide and the big 3: Valproate, carbamazepine, and phenytoin). Side effects are important, but again, broadly:
-Phenytoin: Fetal hydantoin syndrome, folate deficiency
-Carbamazepine: Neutropenia
-Valproate: Pancreatitis, hirsutism
-Others in general: Steven-Johnson Syndrome
Indications are also important. Besides the big 3 again, must-know indications are ethosuxamide for absence seizures and MgSO4 for eclamptic seizures.
All in all, you'll see what's ultimately necessary when you do UWorld and NBME's.
-Phenytoin: Fetal hydantoin syndrome, folate deficiency
-Carbamazepine: Neutropenia
-Valproate: Pancreatitis, hirsutism
-Others in general: Steven-Johnson Syndrome
Indications are also important. Besides the big 3 again, must-know indications are ethosuxamide for absence seizures and MgSO4 for eclamptic seizures.
All in all, you'll see what's ultimately necessary when you do UWorld and NBME's.
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thanks, im about to start my neuro ?'s, also, when it comes to the questoins on NBME, does it really signify the particular topics they ask/dont ask, or it it more about the style of questions that is most important for the nbmes.
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I'd say a bit of both. Certainly after doing UWorld + NBME's together, you'll have a feel for what is and isn't important.
After going through a decent amount of your dedicated prep when you're going back to pick up the scraps (e.g. what you may have missed in your first pass of things), you may consider trying to memorize the entire table, but there are just so many more important things to learn before that.
After going through a decent amount of your dedicated prep when you're going back to pick up the scraps (e.g. what you may have missed in your first pass of things), you may consider trying to memorize the entire table, but there are just so many more important things to learn before that.
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You'll figure out ways to organize the table based on indications & mechanisms. It sucks at first & you miss a bunch of questions on UW, then you start to get it & NBME Qs are pretty straightforward.
As for NBMEs overall, the style was similar to 95+% of questions on my exam - it was obvious or very clear what they are asking & there was a decent amount of extremely straightforward questions (buzzword/phrase gives the answer away, don't need to read any of the rest of the stem). The content was what was different for the most part. They'll ask you aspects of diseases that you most likely never considered before then, since they aren't in review resources, but most of the time you can reason through them based on general knowledge of the condition, whereas NBMEs asked the classic questions & topics that are repeated in every resource.
As for NBMEs overall, the style was similar to 95+% of questions on my exam - it was obvious or very clear what they are asking & there was a decent amount of extremely straightforward questions (buzzword/phrase gives the answer away, don't need to read any of the rest of the stem). The content was what was different for the most part. They'll ask you aspects of diseases that you most likely never considered before then, since they aren't in review resources, but most of the time you can reason through them based on general knowledge of the condition, whereas NBMEs asked the classic questions & topics that are repeated in every resource.
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^yeah that would seem how they'd do it, everyone has access to the resources, so they want to see who can reason and who can't. btw not sure if i should ask this here, but besides the substrates of anti-epileptics, theophylinne warfarin and OCP's shoudl we know any other HY substrates that also are acted upon by the cp450 system
