cardiogenic pulmonary edema physiology question

[ijn]

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What happens to interstitial oncotic pressure when you have pulmonary edema secondary to a myocardial infarction?

 

[Phloston]

What happens to interstitial oncotic pressure when you have pulmonary edema secondary to a myocardial infarction?

It stays the same.

The one notable time that interstitial oncotic pressure increases is with lymphatic obstruction.

With pulmonary oedema secondary to MI, there's just increased capillary hydrostatic pressure, leading to transcudation.

Hydrostatic pressure = osmotic pressure, which is different from oncotic pressure.

 

[banister]

Hydrostatic pressure = osmotic pressure, which is different from oncotic pressure.

Hydrostatic pressure does not = osmotic pressure.

Oncotic pressure = osmotic pressure due to a solute

For our intents and purposes, oncotic ~ osmotic pressure...

 

[Phloston]

Hydrostatic pressure does not = osmotic pressure.

Oncotic pressure = osmotic pressure due to a solute

For our intents and purposes, oncotic ~ osmotic pressure...

I really don't know what you're in disagreement with.

 

[thinkenergy]

I really don't know what you're in disagreement with.

Pulmonary interstitial oncotic pressure actually decreases, b/c increased capillary filtration washes out interstitial proteins, reducing protein concentration. The same reason for why you have increased capillary oncotic pressure at the beginning of renal efferent arteriole.

 

[usmlesuccess]

thinkenergy is right. When you have transudation, there is washout of proteins, which dilutes the proteins and decreases the oncotic pressure in the interstitium. Phloston is wrong.