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Let's say in a situation where a patient has a severe CHF exacerbation where they are third-spacing water everywhere, their RAAS is running in full gear and they're retaining fluids to the point of volume overload. Common sense says we diurese them for symptomatic relief and give them some inotropic support.
Conceptually this makes sense to me. However, what I'm trying to grasp here is the idea that even though they might have more TBW on board, what does that say about their intravascular fluid volume?
Basically, I guess what I'm asking is should we expect patients with CHF exacerbations to be volume overloaded and diurese, leaving fluid resuscitation only if/when they are showing signs of volume depletion with hypotension and tachycardia?
Conceptually this makes sense to me. However, what I'm trying to grasp here is the idea that even though they might have more TBW on board, what does that say about their intravascular fluid volume?
Basically, I guess what I'm asking is should we expect patients with CHF exacerbations to be volume overloaded and diurese, leaving fluid resuscitation only if/when they are showing signs of volume depletion with hypotension and tachycardia?