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nbc

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Plz help with these Q's!!

What lines the non articulating surface of the TMJ?

How does Fluordie affect the shape of the Hydroxyapatite crystals?


What is mutually compromised occlusion?

I got these Questions for my NBDE 1.

Thnx
 
The non-articulating surface of TMJ is lined by the periosteum..

Was that mutually compromised occlusion or mutually protected occlusion??
 
anaesthesia said:
The non-articulating surface of TMJ is lined by the periosteum..

Was that mutually compromised occlusion or mutually protected occlusion??

hi

am sorry .It was mutually protected occlusion.

There was no periosteum in the choice.
It was
fibrocartilage,endothelial cells,synovial lining ,hyaline cartilage.
So wil the answer be endothelial lining???
 
nbc said:
hi

am sorry .It was mutually protected occlusion.

There was no periosteum in the choice.
It was
fibrocartilage,endothelial cells,synovial lining ,hyaline cartilage.
So wil the answer be endothelial lining???

anyone guys?????
 
For the non articulating surface it,s synovial membrane cartilage but not sure.
 
Then it's synovial membrane, it lines the nonarticular portions and the intra-articular ligaments.
Endothelium lines the cardiovascular and lymph vessels.

A mutually protected occlusion includes a lower jaw that is comfortably
seated in the joint and there is even simultaneous contact of the back teeth and light contact of the front teeth. There should also be anterior guidance, cuspid disclussion, and the elimination of balancing side interferences to maintain good health of the teeth, muscles and joints (TMJ).
Mutually protected occlusion assumes that the posterior teeth are most effective for stopping mandibular closure because they are positioned to receive these forces along their long axes.
 
anaesthesia said:
Then it's synovial membrane, it lines the nonarticular portions and the intra-articular ligaments.
Endothelium lines the cardiovascular and lymph vessels.

A mutually protected occlusion includes a lower jaw that is comfortably
seated in the joint and there is even simultaneous contact of the back teeth and light contact of the front teeth. There should also be anterior guidance, cuspid disclussion, and the elimination of balancing side interferences to maintain good health of the teeth, muscles and joints (TMJ).
Mutually protected occlusion assumes that the posterior teeth are most effective for stopping mandibular closure because they are positioned to receive these forces along their long axes.


Thankx for ur response.

I have one more question!

When is the act of swallowing the maximum?

sleep/rest
chewing
during the day

I dont remember the last choice!!!
 
I think it's during the day..
Frequency of swallowing is high while we are awake, almost double in between meals than during eating.. the lower levels of salivary flow during sleep result in less need to swallow

I hope it clears your doubt.. what are the other questions you got in your exam plz let us know..
 
nbc said:
Thankx for ur response.

I have one more question!

When is the act of swallowing the maximum?

sleep/rest
chewing
during the day

ANs is day time.1/3rd occurs during eating,1/12th during sleep and rest balanced during waking/day time.
 
ridge said:
nbc said:
Thankx for ur response.

I have one more question!

When is the act of swallowing the maximum?

sleep/rest
chewing
during the day

ANs is day time.1/3rd occurs during eating,1/12th during sleep and rest balanced during waking/day time.
Definetely, during the day !
jojo-z
🙂
 
1)An important early event in the chemotactic process is cellular adherence to vascular endothelium. The upregulation of endothelial adhesion molecules(ICAM-1 and VCAM-1)is part a response to

a. cytokine stimulation
b. lysozyme production
c. mast cell degranulation
d. antibody binding to endothelial Fc receptors
e. protease inhibition by serum anti-proteases
 
1] In a primary mandibular first molar which is least in height?

mesial
midfacial
midlingual
distal

2] in a mutually protected occlusion what happens when the right maxillary canine is extracted ?
I dont remember the choices...

The choices had the effects on posterior teeth/anterior teeth and 2 more choices.

3]Can someone plz explain what is canine guidance occlusion and what happens when a canine is absent/missing in the left maxillary quadrant.How does it affect the movement.

4]Which is the teeth to calcify at the oldest age?

There was no 3rd molar in the choice.

Thankx
 
nbc said:
hi

am sorry .It was mutually protected occlusion.

There was no periosteum in the choice.
It was
fibrocartilage,endothelial cells,synovial lining ,hyaline cartilage.
So wil the answer be endothelial lining???

The non-artriculating TMJ is lined by fibrocartilage while the artriculating area is covered by synovial lining.
 
hzq838 said:
The non-artriculating TMJ is lined by fibrocartilage while the artriculating area is covered by synovial lining.

1] In a primary mandibular first molar which is least in height?

mesial
midfacial
midlingual
distal

2] in a mutually protected occlusion what happens when the right maxillary canine is extracted ?
I dont remember the choices...

The choices had the effects on posterior teeth/anterior teeth and 2 more choices.

3]Can someone plz explain what is canine guidance occlusion and what happens when a canine is absent/missing in the left maxillary quadrant.How does it affect the movement.

4]Which is the teeth to calcify at the oldest age?

There was no 3rd molar in the choice.

Thankx

Still waiting!!!Anyone!
 
Well i don't know how to explain this in a easy way but i'm gonna try my best....

Now let's try and explain how an individual with an ideal bite functions. Starting at centric Occlusion and keeping the teeth in contact as much as possible, we slide the lower jaw to one side. In an ideal Occlusion, when the lower jaw is shifted to one side, the posterior teeth should not touch.

What happens is that the lower canine is riding up on the upper canine. This is called Canine Guidance. fewer muscles are active when canine contact during eccentric movement than when posterior teeth contact.. So if the canine guidance is absent the muscle stays active, and you can get clenching, grinding of the teeth, joint pain, fracturing of teeth, excessive wear of the enamel on top of the tooth...

The best alternative for canine guidance is group function when several teeth contact on the working side during the laterotrusive movement... like canine, premolas n 1st molar....

In mutually protected occlusion there should be anterior guidance n canine disclussion n elimination of balancing side interference.... so i guess if canine guidance is lost there will be balancing side interference???????

Sorry i really don't know the exact answer to your question... but i do hope now you know what is canine guidance and mutually protected occlusion..
 
anaesthesia said:
Well i don't know how to explain this in a easy way but i'm gonna try my best....

Now let's try and explain how an individual with an ideal bite functions. Starting at centric Occlusion and keeping the teeth in contact as much as possible, we slide the lower jaw to one side. In an ideal Occlusion, when the lower jaw is shifted to one side, the posterior teeth should not touch.

What happens is that the lower canine is riding up on the upper canine. This is called Canine Guidance. fewer muscles are active when canine contact during eccentric movement than when posterior teeth contact.. So if the canine guidance is absent the muscle stays active, and you can get clenching, grinding of the teeth, joint pain, fracturing of teeth, excessive wear of the enamel on top of the tooth...

The best alternative for canine guidance is group function when several teeth contact on the working side during the laterotrusive movement... like canine, premolas n 1st molar....

In mutually protected occlusion there should be anterior guidance n canine disclussion n elimination of balancing side interference.... so i guess if canine guidance is lost there will be balancing side interference???????

Sorry i really don't know the exact answer to your question... but i do hope now you know what is canine guidance and mutually protected occlusion..

Thnkx a lot Anaesthesia.I suppose ur explanation will surely help.Will try to find the answer.
Thanks again.
 
nbc said:
1] In a primary mandibular first molar which is least in height?

mesial
midfacial
midlingual
distal

2] in a mutually protected occlusion what happens when the right maxillary canine is extracted ?
I dont remember the choices...

The choices had the effects on posterior teeth/anterior teeth and 2 more choices.

3]Can someone plz explain what is canine guidance occlusion and what happens when a canine is absent/missing in the left maxillary quadrant.How does it affect the movement.

4]Which is the teeth to calcify at the oldest age?

There was no 3rd molar in the choice.

Thankx

In case of a primary mand 1st molar, mesiolingual cusp is the largest(same as in permanent 1st max molar) and the distolingual cusp is the smallest...so that wayz the answer should be distolingual cusp but am nt sure
 
drkum1 said:
1)An important early event in the chemotactic process is cellular adherence to vascular endothelium. The upregulation of endothelial adhesion molecules(ICAM-1 and VCAM-1)is part a response to

a. cytokine stimulation
b. lysozyme production
c. mast cell degranulation
d. antibody binding to endothelial Fc receptors
e. protease inhibition by serum anti-proteases

I think the answer is cytokine stimulation
 
nbc said:
1] In a primary mandibular first molar which is least in height?

mesial
midfacial
midlingual
distal

2] in a mutually protected occlusion what happens when the right maxillary canine is extracted ?
I dont remember the choices...

The choices had the effects on posterior teeth/anterior teeth and 2 more choices.

3]Can someone plz explain what is canine guidance occlusion and what happens when a canine is absent/missing in the left maxillary quadrant.How




does it affect the movement.

















4]Which is the teeth to calcify at the oldest age?

There was no 3rd molar in the choice.

Thankx

Still waiting!!!Anyone!



what were the other choices for the 4th question?
 
nbc said:
Plz help with these Q's!!

What lines the non articulating surface of the TMJ?

How does Fluordie affect the shape of the Hydroxyapatite crystals?


What is mutually compromised occlusion?

I got these Questions for my NBDE 1.

Thnx


When did you give your exam?Please let us know more questions if u remember from all the subjects...do u have question papers frm 1999-2005?
 
Thank you Mahima. Can you explain...
[
QUOTE=mahima_dentist]I think the answer is cytokine stimulation[/QUOTE]
 
drkum1 said:
Thank you Mahima. Can you explain...
[
QUOTE=mahima_dentist]I think the answer is cytokine stimulation
[/QUOTE]

ICAM(Intercellular cell adhesion molecule ) and VCAM(vascular cell adhesion molecule) are present in the endothelial cells..and the interleukins(which are a type of cytokines) like IL1,TNF(Tumour necrosis factor) increase the endothelial cell adhesiveness for leukocytes in chemotaxis(during inflammation)by stimulating the surface expression of various adhesion molecules (the ones mentioned above) thereby attracting the leukocytes in increased quantities and letting them move to the site of inflammation by diapedesis...thats all what i know
 

ICAM(Intercellular cell adhesion molecule ) and VCAM(vascular cell adhesion molecule) are present in the endothelial cells..and the interleukins(which are a type of cytokines) like IL1,TNF(Tumour necrosis factor) increase the endothelial cell adhesiveness for leukocytes in chemotaxis(during inflammation)by stimulating the surface expression of various adhesion molecules (the ones mentioned above) thereby attracting the leukocytes in increased quantities and letting them move to the site of inflammation by diapedesis...thats all what i know[/QUOTE]


When did you give your exam?Please let us know more questions if u remember from all the subjects...do u have question papers frm 1999-2005?
 
[QUOTE=nbc
What lines the non articulating surface of the TMJ?
ANS IS SYNOVIAL LINING AS ARTICULAR SURFACE IS LINED BY FIBROUS CONNECTIVE TISSUE.

How does Fluordie affect the shape of the Hydroxyapatite crystals?
ans:makes it more stronger to resist dissolution
any choices????????? for this question
 
Flouride turns hydroxyapatite to fluoroapatite i.e. Ca ion is replaced by fluoride ion, which makes it more resistant to acid penetration.

Order of calcification: Mand 1molar-max 1 molar- mand central- max central- mand lateral- mand canine- max lateral- max canine- max pm1- mand pm1- max pm2- mand pm2- max m2- mand m2- max m3- mand m3


plz Correct me if wrong




 
Kshah said:
Flouride turns hydroxyapatite to fluoroapatite i.e. Ca ion is replaced by fluoride ion, which makes it more resistant to acid penetration.
Correct me if wrong




The question asked about change in shape!!Rather if you cud explain how the Fl is integrated into the hydroxyapatite crystal.
 
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