Hi, I do not know any webpage that summarizes those in a high yield manner for step1. For concrete information about them you should consult an internal medicine book like Harrison's. But let me try to shed some light on them:
wheezes: You hear them in obstructive diseases. Its produced when the caliber of the bronchioles are reduced, so the air flow is turbulent and difficult, and you hear it as a wheeze. When the obstruction is mild, it will start with only during expiration, when it is moderate, you will hear it on expiration and inspiration, then only in inspiration. This is explained because during expiration the caliber of the bronchiles is reduced, and in inspiration, the caliber is increased, so even with a mild obstruction, the expiratory phase will be the first one compromised, and as it gets worse, it will even start compromising the inspiratory phase where the bronchioles should be at its largest caliber. And when the obstruction is so severe you won't hear anything, an emergency condition called silent thorax. The obstruction is so severe that no air can get out. Some students will auscultate, hear nothing and think that the patient is alright, when in reality he is suffocating.
To continue with the obstructive diseases. Hyper-resonant refers to the thorax that is filled with air. Like when you see it in an xray, it is hyperinflated, completely hypodense (looks black), its explained because there is air trapping, like in COPD or asthma. So when you tap with your fingers on the thorax, it sounds like a hollow barrel. It can also be seen in pneumothorax.
Compare and contrast with other conditions, like pneumonia, or pleural effusion, where the lungs and its cavities are filled with something. And when you tap, it will be hyporesonant.
Ronchi are heard when there is fluid in the alveolar space. Happens in different conditions, like pneumonia, or CHF. It is produced because of the inflation and deflation of alveoli, and the mobiilization of this fluid. If you want an example of that sound, grab your hair that is closest to your ear, and rub it against each other. That sounds like ronchi.
In the same conditions, tactile fremitus is produced when you put your hand on top of the chest wall of the patient, make him do inspirations and expirations, and if you feel a slight vibration during those, it means the alvoli are filled with fluid. Basically it is the ronchi, but felt during tactile examination.
Another important sign is aegophony. You make the patient say out loud letters like "eeeee". In a normal patient, while auscultating the chest, you shouldnt be able to hear this letters so clear. But in a patient that has some disease like pnuemonia, or CHF, or fibrosis, where there is fluid or something filling the alvoli and producing a consolidating lesion, you can hear the "eeeee" as though the patient was talking right next to your ear. It is produced because in consolidating lesions, the sound vibrations travel much better than in an air filled space. You can test this with a stethoscope, or even with your hands on the chest to sense the vibrations.
So to recap:
Wheezes, decreased tactile fremitus, decreased aegophony are found on pulmonary obstructions. Most common, asthma, COPD, anaphylaxis, allergies.
Ronchi, tactile fremitus, aegophony are found on consolidating lesions, like pneumonia, CHF, aspiration.
Hyper-resonant thorax -> Obstructive diseases AND pneumothorax
Hyporesonant thorax -> consolidating diseases AND pleural effusion
Hope it helps. But I would still check Harrison's.
