NBME FORM 4 QUESTION about alveoli

[Ihateverbal]

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There was some kind of question on NBME Form 4 with a histo picture of alveolar (type I pneumocyte) wall and then it asked what kind of disease woudl MOST effect this.

The answer choices were something along the line of like asbestosis, silicosis, asthma, etc...

Does anyone remember what the put and why? I thought it would be a restrictive disease so it woudl be asbestosis/silicosis but I think either could cause fibrosis. So I assumed I must have gotten it wrong and it was probably none of those choices

 

[NCF145]

There was some kind of question on NBME Form 4 with a histo picture of alveolar (type I pneumocyte) wall and then it asked what kind of disease woudl MOST effect this.

The answer choices were something along the line of like asbestosis, silicosis, asthma, etc...

Does anyone remember what the put and why? I thought it would be a restrictive disease so it woudl be asbestosis/silicosis but I think either could cause fibrosis. So I assumed I must have gotten it wrong and it was probably none of those choices

No option for emphysema?

 

[Ihateverbal]

no option for emphysema (from what I remember)

 

[Zweisamkeit]

No option for emphysema?

Nope, no emphysema.

I was tripped up on this question too. It was definitely a picture of aveoli. But I thought asbestosis and silicosis could both affect the aveoli. 😕

 

[Ihateverbal]

that's what I thought...I guessed and put silicosis, only because when I think of asbestos, I think more of mesothelioma 😀

But I scored really well on respiratory (and even got a little asterick on the right hand side of my bar) so I'm wondering if I got it right...who knows?

 

[SeminoleFan3]

Just took it today...one of the options was lobar consolidation, which is what I put because it causes intra-alveolar exudate.

 

[TMP-SMX]

All of the fibrotic diseases are more interstitial by definition (connective tissue). Pneumonia involves infection of the alveoli themselves and filling with exudate. Cavitary lesions are destruction of the alveoli.

 

[Ihateverbal]

i see where you are coming from...

oh well...i knew it couldn't have been an interstitial problem since two of the 5 answer choices were interstitial.

 

[Mortal_Lessons]

I have a question about a concept that was tested on NBME form 5, so please do not look ahead if you are planning on doing this form.













In an experiment, if you decrease the oxygen concentration of inspired air that someone is breathing, there won't be much of an effect on their respiratory drive until you reach a critical pO2 where there'll be an exponential rise in respiratory effort. Until that point, it's the pC02/H+ that really governs respiratory drive in the carotid bodies and central chemoreceptors. My question has to do with the removal of the carotid bodies and its effect on respiratory drive. If you now begin to decrease the oxygen concentration of inspired air, will the respiratory drive continue to be the same at any concentration? I was looking at some answers online and one key suggests that the respiratory drive will continue to increase at a steady rate as soon as you begin decreasing oxygen concentration-that kind of answer seems to imply there's a concomitant change in pCO2 when you decrease the p02. When you decrease p02, doesn't pCO2 continue to be the same?

 

[ENThopeful]

while we are asking questions on form 5,





what was that baby's thumb affected with? it was some virus but i couldnt figure out what the heck it was.

 

[Mortal_Lessons]

while we are asking questions on form 5,





what was that baby's thumb affected with? it was some virus but i couldnt figure out what the heck it was.

Based on the available answer choices, I figured it was hand-foot-mouth disease due to coxsackie A virus (+ss RNA unenveloped). I really had to stretch to think of that one-at first I wanted to say it was some kind of DNA virus (like a herpesvirus) but that choice wasn't available.

Any thoughts on the above question about removing the carotid bodies and observing the effects of a decreasing oxygen concentration on respiratory drive?