When you're thinking about this stuff, the first thing you need to start with is knowing what the Albright hereditary osteodystrophy (AHO) phenotype is.
Albright hereditary osteodystrophy = the clinical presentation of shortened fourth/fifth metacarpals, short stature, mild mental ******ation and round facies. That description is essentially the common tetrad you'll hear.
The second step is looking at the biochemistries. If calcium is low, phosphate high, and PTH high, then there's PTH insensitivity. We can call this biochemistry pseudohypoparathyroidism (Ca/PO4 reflect as if PTH is low, but it's actually high)
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AHO phenotype + pseudo biochemistry = pseudohypoparathyroidism type Ia
AHO phenotype + normal biochemistry = pseudopseudohypoparathyroidism (phenotype looks like pseudo Ia but biochemistries say otherwise)
Normal phenotype + pseudo biochemistry = pseudohypoparathyroidism Ib or II (in II the defect is more downstream and cAMP responses are normal in response to exogenous PTH)
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The USMLE will tell you there's pseudohypoparathyroidism and then ask for what happens when exogenous PTH is given. Normally nothing should occur because the disease is as if PTH isn't there. But if you're cornered into a situation where something has to respond, it's urinary cAMP, which will increase in type II.