Hello friends,
I got a question wrong and i'm trying to figure out how afferent and efferent arterioles change their sm. muscle tone during hypovolemia.
What I do know: During hypovolemia, both afferent and efferent arterioles constrict.
What I want to know: How does it get there?
One question in particular: I know angII causes efferent constriction preferentially over afferent. FA says that JG cells (in the afferent arteriole) maintains GFR via RAAS. How do I reconcile these two facts?
Thanks!
I got a question wrong and i'm trying to figure out how afferent and efferent arterioles change their sm. muscle tone during hypovolemia.
What I do know: During hypovolemia, both afferent and efferent arterioles constrict.
What I want to know: How does it get there?
One question in particular: I know angII causes efferent constriction preferentially over afferent. FA says that JG cells (in the afferent arteriole) maintains GFR via RAAS. How do I reconcile these two facts?
Thanks!