it is and infection to lung parenchyma, rite?
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what do u know about pneumonia?
Started by freakystud
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how is cause hardening to the lung? anyone know any pathophysiology that lead to hardening?
from what i know, pneumonia result whenever pulmonary defence mechanism impaired like in HIV/AIDS patients or immunosuppred patients (eg. in chemotherapy, pregnancy, any steroid drugs) .commonly in HIV its the pneumonia is caused by fungi called pneumocyctis jirovecii..( PCP). but still pneumonia caused by variesties og agents including bacterias such as pneumococci,klebsiella,streptococci,staphylococci, haemophilus influennza,psedomonas aeruginosa.if there is any mistakes,plz do correct me...
from what i know, pneumonia result whenever pulmonary defence mechanism impaired like in HIV/AIDS patients or immunosuppred patients (eg. in chemotherapy, pregnancy, any steroid drugs) .commonly in HIV its the pneumonia is caused by fungi called pneumocyctis jirovecii..( PCP). but still pneumonia caused by variesties og agents including bacterias such as pneumococci,klebsiella,streptococci,staphylococci, haemophilus influennza,psedomonas aeruginosa.if there is any mistakes,plz do correct me...
Maybe your spell check exploded, but you get hardening in pneumonia due to consolidation. This is because there is destruction of the normal architecture of the lung and have inflammation and solidified pus instead. Acute pneumonia can turn into chronic pneumonia and as is the case with chronic bronchitis (obviously more bronchitis than pneumonia but same deal) or cystic fibrosis (bronchiectasis). Chronic inflammation includes fibrosis. Still, anyone can get bacterial pneumonia. Depending on the age group the organisms vary and in certain predisposed individuals can get organisms more than normal.
CF=Pseudomonas (Though the common organisms like S. aureus are still more common but they are usually on long-term prophylactic treatment for S. aureus and tend to get highly resistant Pseudomonas strains)
Alcoholic (Klebsiella and red currant jelly sputum though like I said others are still more common). Aspiration pneumonia is also more common and usually polymicrobial with anaerobes.
Sickle cell (or anyone without a functioning spleen)=Encapsulated organisms
AIDS=Fungi/TB (though anything is possible)
Strep pmeumo is still one of the more common organisms and causes rusty colored sputum
FYI, pneumococcus is the same as strep pneumo in case you didn't know as you listed it twice.
CF=Pseudomonas (Though the common organisms like S. aureus are still more common but they are usually on long-term prophylactic treatment for S. aureus and tend to get highly resistant Pseudomonas strains)
Alcoholic (Klebsiella and red currant jelly sputum though like I said others are still more common). Aspiration pneumonia is also more common and usually polymicrobial with anaerobes.
Sickle cell (or anyone without a functioning spleen)=Encapsulated organisms
AIDS=Fungi/TB (though anything is possible)
Strep pmeumo is still one of the more common organisms and causes rusty colored sputum
FYI, pneumococcus is the same as strep pneumo in case you didn't know as you listed it twice.
is cystic fibrosis same with bronchiectasis??
i thought bronchiectasis is permanent dilatation of the airway.
i thought bronchiectasis is permanent dilatation of the airway.
how is cause hardening to the lung? anyone know any pathophysiology that lead to hardening?
from what i know, pneumonia result whenever pulmonary defence mechanism impaired like in HIV/AIDS patients or immunosuppred patients (eg. in chemotherapy, pregnancy, any steroid drugs) .commonly in HIV its the pneumonia is caused by fungi called pneumocyctis jirovecii..( PCP). but still pneumonia caused by variesties og agents including bacterias such as pneumococci,klebsiella,streptococci,staphylococci, haemophilus influennza,psedomonas aeruginosa.if there is any mistakes,plz do correct me...
how does it cause hardening to the lung? Does anyone know any pathophysiology that leads to hardening? As far as I know, pneumonia occurs whenever pulmonary defense mechanisms are impaired, like with HIV/AIDS patients, or immunosuppresed patients (eg. with chemotherapy, pregnancy, any steroid drugs). Commonly in HIV the pneumonia is caused by fungi called pneumocyctis jirovecii..( PCP). but still pneumonia is caused by various agents including bacteria such as pneumococci, klebsiella, streptococci, staphylococci, haemophilus influenza, psedomonas aeruginosa. If there are any mistakes, please do correct me.
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is cystic fibrosis same with bronchiectasis??
i thought bronchiectasis is permanent dilatation of the airway.
It isn't the same, but many CF patients (or chronic bronchitis patients) tend to get it because of recurrent infections of the bronchi. You get a hardened and inflamed airway full of mucus.
The guy obviously can't speak english very well, but anyone can easily decipher what he is trying to say without your condescending, "translation."
Hey man he asked to be corrected.
Maybe your spell check exploded, but you get hardening in pneumonia due to consolidation. This is because there is destruction of the normal architecture of the lung and have inflammation and solidified pus instead. Acute pneumonia can turn into chronic pneumonia and as is the case with chronic bronchitis (obviously more bronchitis than pneumonia but same deal) or cystic fibrosis (bronchiectasis). Chronic inflammation includes fibrosis. Still, anyone can get bacterial pneumonia. Depending on the age group the organisms vary and in certain predisposed individuals can get organisms more than normal.
CF=Pseudomonas (Though the common organisms like S. aureus are still more common but they are usually on long-term prophylactic treatment for S. aureus and tend to get highly resistant Pseudomonas strains)
Alcoholic (Klebsiella and red currant jelly sputum though like I said others are still more common). Aspiration pneumonia is also more common and usually polymicrobial with anaerobes.
Sickle cell (or anyone without a functioning spleen)=Encapsulated organisms
AIDS=Fungi/TB (though anything is possible)
Strep pmeumo is still one of the more common organisms and causes rusty colored sputum
FYI, pneumococcus is the same as strep pneumo in case you didn't know as you listed it twice.
Not sure what you mean there by chronic pneumonia? Are you talking about interstital lung diseases or maybe airway scarring in the upper lungs seen in X-Rays patients who once had TB? I'm also not sure what the OP means bt hardenining, but that may simply be due to where i'm learning it (since I do know pna itself isn't a universal term)
PS, just an addendum/cool anecdote since. I've only dx'd one pna based on sputum color. The rest never had the typical colors i've read about. But it was really cool. There was bright lime fluorescent green on a pateint with ventilator associated pna. yep. Pseudomonas🙂
Any chronic condition will lead to fibrosis (especially in tissues that are slightly more prone to fibrosis as is the lung)...so any chronic pneumonia (due to Immunocompromised, malnuritioned, elderly, smokers...etc) will lead to fibrosis over time.
BUT INTERSTITIAL PNEUMONIAs ( the only agents I can think of at the moment that causes interstitial pneumonia is Mycoplasma Peumoniae, Viral Peumonias (atypical pneumonias = effect septa), and Pneumocystis..) --> bc they directly infect the septal cells they are very likely to lead to Fibrosis in a quicker amount of time. You will actually see very significantly enlarged alveolar septa in slides.
Thats what I got after finishing respiratory path this past week👍
Please correct me if I m wrong or add more (will help me also for my exam in 3 weeks..😀).
BUT INTERSTITIAL PNEUMONIAs ( the only agents I can think of at the moment that causes interstitial pneumonia is Mycoplasma Peumoniae, Viral Peumonias (atypical pneumonias = effect septa), and Pneumocystis..) --> bc they directly infect the septal cells they are very likely to lead to Fibrosis in a quicker amount of time. You will actually see very significantly enlarged alveolar septa in slides.
Thats what I got after finishing respiratory path this past week👍
Please correct me if I m wrong or add more (will help me also for my exam in 3 weeks..😀).