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BRS (page 33 ques 8) has a question where it says that in a HIV + patient with TUberculosis, epitheliod cells are an invariable feature.
Goljan in his audio lectures mentions that in HIV, helper T cells are so depleted that there is absoultely no chance of granuloma formation (reason : macrophages are not activated). as they aren't activated how does one INVARIABLY get epitheliod cells?
Goljan in his audio lectures mentions that in HIV, helper T cells are so depleted that there is absoultely no chance of granuloma formation (reason : macrophages are not activated). as they aren't activated how does one INVARIABLY get epitheliod cells?