Anatomy question

This forum made possible through the generous support of SDN members, donors, and sponsors. Thank you.
Basicly, adjust the blood flow to lacrimal gland via contracting arterioles(alpha receptor). some researcher also found Sympathetic N. also control tear secrtion along with Parasympathetic N.
 
The MOST susceptible sites in the nervous system for the effects of acute ischemic anoxia are the
A. motor end-plates.
B. sensory receptors.
C. synapses on autonomic ganglia.
D. synapses in the central nervous system.


 
the pt complains of pain at the injection site and inability 2 open his mouth after the restorative treatment on 19 .this is due to
a]spasm of temporalis muscle
b] inj into medial pterigoid muscle
c] trauma to inferior alveolar nerve
d] damage to facial nerve
 
The MOST susceptible sites in the nervous system for the effects of acute ischemic anoxia are the
A. motor end-plates.
B. sensory receptors.
C. synapses on autonomic ganglia.
D. synapses in the central nervous system.




my guess is c, because resipiration is controlled involuntarily
 
the pt complains of pain at the injection site and inability 2 open his mouth after the restorative treatment on 19 .this is due to
a]spasm of temporalis muscle
b] inj into medial pterigoid muscle
c] trauma to inferior alveolar nerve
d] damage to facial nerve

Answer is B. If you inject too far medially you can hit medial pterygoid and get guarding trismus.
 
the pt complains of pain at the injection site and inability 2 open his mouth after the restorative treatment on 19 .this is due to
a]spasm of temporalis muscle
b] inj into medial pterigoid muscle
c] trauma to inferior alveolar nerve
d] damage to facial nerve

i think it would be d because the facial nerve also innervates some of the suprahyoid muscle that aids the lateral pterygoid in depressing the mandible
 
The MOST susceptible sites in the nervous system for the effects of acute ischemic anoxia are the
A. motor end-plates.
B. sensory receptors.
C. synapses on autonomic ganglia.
D. synapses in the central nervous system.



or it could be d since hypoixia for an amount of time can cause permenant brain damage
 
i think it would be d because the facial nerve also innervates some of the suprahyoid muscle that aids the lateral pterygoid in depressing the mandible

decent thought but trust me its medial pterygoid. its happened to people in our class when we were learning injections on one another 😀
===================
Trismus (http://www.novocol.com/Septodont/english/other/cea_dh01.html)

Trismus, or limited jaw opening, is relatively common following an injection of local anaesthetic. It can be due to needle insertion into one of the muscles of mastication, leading to bleeding, spasm or both. The muscle most commonly involved is the medial pterygoid, which can be penetrated during an inferior alveolar nerve block using any of the three main techniques: the conventional approach, the Vazirani-Akinosi technique or the Gow-Gates. Less commonly, the temporalis may be penetrated near its attachment onto the coronoid process if the needle is inserted too far laterally or superiorly. Rarely, the lateral pterygoid muscle may be penetrated if a block is administered too far superiorly.

Injection of local anaesthetic directly into muscle may cause a mild myotoxic response, which can lead to necrosis.

The symptoms of trismus, often associated with pain, arise anywhere from 1 to 6 days following an injection. The duration of symptoms and their severity are both variable. With management as described below, improvement should be noted within 2 to 3 days. If there is no improvement within this time, the dentist should consider other possible causes (e.g., infection) and treat accordingly.
 
Top