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Asymptomatic HTN
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There should only be either hypertensive emergency or asymptomatic hypertension. There's no in between. I'm glad they got rid of the "hypertensive urgency" language but they should go further.
Could petition AHA to rename hypertensive urgency to vascular fibromyalgia
Sincere question - In your opinion(s) if PCP office lab can’t get lab results for a few days because of send out process, would that support sending to higher level of care, a la ED?There is now an AHA Scientific Statement AGAINST acutely treating asymptomatic hypertension.
Thus, there should no need for anyone to educate other physicians. Simply ask for their email, attach this article, and tell them that you are following guidelines from the authoritative professional society on the matter.
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I’ve become more accommodating with HTN in the ED.
Most people are asymptomatic. Anxious. It doesn’t hurt much to pull and EKG and or BMp
And then talk to them like a human. Explain there are downsides to rapid treatment. The best thing is to see their PCP in a week or two, see if they are riding high, and decide on tx.
Now there is a significant subset when you see them they are 200/100. They’ve been told they have HTN for years. They’ve avoided treatment (usually these are men in their 40s-50s). For some reasons they Checked BP today. Their wife : GF drug them in
I have zero issue starting them on a single agent. But they also most benefit from rapid follow up.
Most people are asymptomatic. Anxious. It doesn’t hurt much to pull and EKG and or BMp
And then talk to them like a human. Explain there are downsides to rapid treatment. The best thing is to see their PCP in a week or two, see if they are riding high, and decide on tx.
Now there is a significant subset when you see them they are 200/100. They’ve been told they have HTN for years. They’ve avoided treatment (usually these are men in their 40s-50s). For some reasons they Checked BP today. Their wife : GF drug them in
I have zero issue starting them on a single agent. But they also most benefit from rapid follow up.
Sincere question - In your opinion(s) if PCP office lab can’t get lab results for a few days because of send out process, would that support sending to higher level of care, a la ED?
No.
Asymptomatic HTN is asymptomatic HTN.
They -will- tell you that they have some symptom. Ignore that.
That AHA paper says BMP to assess for end organ dysfunction (kidneys), EKG (heart), fundoscopic exam (retina).No. Asymptomatic HTN is asymptomatic HTN.
They -will- tell you that they have some symptom. Ignore that.
So what you’re saying is don’t follow evidence & recs. Just enjoy the inevitable malpractice suit that will come their way.
Yup, makes sense.

That AHA paper says BMP to assess for end organ dysfunction (kidneys), EKG (heart), fundoscopic exam (retina).
So what you’re saying is don’t follow evidence & recs. Just enjoy the inevitable malpractice suit that will come their way.
Yup, makes sense.![]()
"I just don't feel right, so I checked my blood pressure and..." is what they typically say. I ignore that nonsense.
Cool story."I just don't feel right, so I checked my blood pressure and..." is what they typically say. I ignore that nonsense.
But I’ll just go ahead and disagree with you on this.
That AHA paper says BMP to assess for end organ dysfunction (kidneys), EKG (heart), fundoscopic exam (retina).
So what you’re saying is don’t follow evidence & recs. Just enjoy the inevitable malpractice suit that will come their way.
Yup, makes sense.![]()
This.
We (as a system) have discussed the cost/benefit ratio for buying every ED a retina scanner so we can go down this road.
Ugh.
Either you're symptomatic, or you're not. The worst offenders by far are the nurse triage lines (that aren't run by a nurse) that tell every patient to go to the ED... then the patient gets pissed that you're not doing anything.
We have those in the office for diabetic eye screenings, totally not needed for the ED but pretty cool nonetheless.This.
We (as a system) have discussed the cost/benefit ratio for buying every ED a retina scanner so we can go down this road.
Ugh.
Either you're symptomatic, or you're not. The worst offenders by far are the nurse triage lines (that aren't run by a nurse) that tell every patient to go to the ED... then the patient gets pissed that you're not doing anything.
That AHA paper says BMP to assess for end organ dysfunction (kidneys), EKG (heart), fundoscopic exam (retina).
I propose we have every ophthalmologist buy an EKG machine and POC testing and we just send all these people there.
I propose we have every ophthalmologist buy an EKG machine and POC testing and we just send all these people there.
I like your thinking. But for real, I can do an EKG & a simple fundoscopic exam. That’s why I asked specifically about labs that won’t come back for a couple of days.I propose we have every ophthalmologist buy an EKG machine and POC testing and we just send all these people there.
Because at the end of the day, good PCPs keep people out of the ED by properly managing chronic medical issues. It’s the acute ones that may necessitate a more immediate higher level of care.
not worth the complaint about not taking their bp seriously and how everyone else lowers their blood pressure
I think that it is reasonable in certain, rare circumstances, and the PCP's office might be behaving in accordance with both the AHA paper and the spirit of ACEP's own clinical policy on asymptomatic HTN if they had a concern and couldn't get a timely creatinine. That is to say, the ACEP policy notes that in patients with poor follow-up, a screening creatinine might identify patients in need of admission. That is a Level C recommendation but what you describe effectively falls in that category. Asymptomatic Elevated Blood Pressure.Sincere question - In your opinion(s) if PCP office lab can’t get lab results for a few days because of send out process, would that support sending to higher level of care, a la ED?
So, if a PCP has a particular concern in an asymptomatic patient such as the trajectory of a patient's BP (ie normally normotensive and now suddenly in the AHA "asymptomatic but markedly elevated" category) I will not fault them for sending the patient to the ED for a renal panel / BMP if it cannot be obtained in a timely (ie 24 hrs) manner in a clinic. I will also not fault any EP who sees that patient and says, "Get the frack outta here - ain't nobody got time for that." That is because it is not my practice to routinely or even occasionally screen an asymptomatic hypertensive; I've perhaps done that on my own conscious volition a couple of times in a 20-year career. Nor do I think that we should be picking up the out-patient's lab slack.
Finally, it's important to note that the AHA paper complements the ACEP policy with respect to not attempting to acutely lower the BP in patients without end organ damage. The AHA paper really doesn't help us with identifying patients who should be screened for organ dysfunction. Better yet, the AHA paper doesn't explicitly tell us NOT to screen asymptomatic patients. There is a tone in this thread that an absence of symptoms is sufficient to rule out hypertensive emergencies and I generally believe this to be correct. However, embedded in both the AHA and ACEP polices is this notion of selective screening certain symptomatic patient populations. This seems to generally center around asymptomatic changes in creatinine that would need treatment. I suspect there is some disagreement on prevalence and risk tolerance on this issue of asymptomatic AKIs among the ED and PCP cohorts, and the various professional guidance seems to allow wiggle room on this question.
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Thank you for your reasoned reply!I think that it is reasonable in certain, rare circumstances, and the PCP's office might be behaving in accordance with both the AHA paper and the spirit of ACEP's own clinical policy on asymptomatic HTN if they had a concern and couldn't get a timely creatinine. That is to say, the ACEP policy notes that in patients with poor follow-up, a screening creatinine might identify patients in need of admission. That is a Level C recommendation but what you describe effectively falls in that category. Asymptomatic Elevated Blood Pressure.
So, if a PCP has a particular concern in an asymptomatic patient such as the trajectory of a patient's BP (ie normally normotensive and now suddenly in the AHA "asymptomatic but markedly elevated" category) I will not fault them for sending the patient to the ED for a renal panel / BMP if it cannot be obtained in a timely (ie 24 hrs) manner in a clinic. I will also not fault any EP who sees that patient and says, "Get the frack outta here - ain't nobody got time for that." That is because it is not my practice to routinely or even occasionally screen an asymptomatic hypertensive; I've perhaps done that on my own conscious volition a couple of times in a 20-year career. Nor do I think that we should be picking up the out-patient's lab slack.
Finally, it's important to note that the AHA paper complements the ACEP policy with respect to not attempting to acutely lower the BP in patients without end organ damage. The AHA paper really doesn't help us with identifying patients who should be screened for organ dysfunction. Better yet, the AHA paper doesn't explicitly tell us NOT to screen asymptomatic patients. There is a tone in this thread that an absence of symptoms is sufficient to rule out hypertensive emergencies and I generally believe this to be correct. However, embedded in both the AHA and ACEP polices is this notion of selective screening certain symptomatic patient populations. This seems to generally center around asymptomatic changes in creatinine that would need treatment. I suspect there is some disagreement on prevalence and risk tolerance on this issue of asymptomatic AKIs among the ED and PCP cohorts, and the various professional guidance seems to allow wiggle room on this question.
Cool story.
But I’ll just go ahead and disagree with you on this.
A-okay.
If I worked up every hysterical senior, I'd not have the time to do anything else.
Do a UA. No blood, no protein, probably fineI like your thinking. But for real, I can do an EKG & a simple fundoscopic exam. That’s why I asked specifically about labs that won’t come back for a couple of days.
Because at the end of the day, good PCPs keep people out of the ED by properly managing chronic medical issues. It’s the acute ones that may necessitate a more immediate higher level of care.
Good call - inexpensive w/ quick in-office result. Reasonable enough for folks who just need to finally start a med.Do a UA. No blood, no protein, probably fine
So there's blood and protein, now what? You do a Cr and it's 1.6 with nl lytes and no anion gap. So you... tell the patient to follow up with their PCP. Or maybe admit them to the special "Not a thing" ward where you pile the asymptomatic acute renal failures in with all the asymptomatic R/O DKA patients the clinic sends to us rather than managing their DM.Do a UA. No blood, no protein, probably fine
Look at Mr. Showoff who can get a UA within 4 hours in the ED.Do a UA. No blood, no protein, probably fine
The point @VA Hopeful Dr was making was that we can easily do dipstick UAs in PCP clinic.So there's blood and protein, now what? You do a Cr and it's 1.6 with nl lytes and no anion gap. So you... tell the patient to follow up with their PCP. Or maybe admit them to the special "Not a thing" ward where you pile the asymptomatic acute renal failures in with all the asymptomatic R/O DKA patients the clinic sends to us rather than managing their DM.
The whole point of my question, and subsequent posts, was how to keep patients OUT of the ED while doing the necessary things to evaluate for undifferentiated badness that might require an admission.
No.
Asymptomatic HTN is asymptomatic HTN.
They -will- tell you that they have some symptom. Ignore that.
That's key.
If they look comfortable, there is no medical emergency.
Do a UA. No blood, no protein, probably fine
Even then, I spoke to a Nephrologist once because I had a pt with asymptomatic HTN with one of those two (I can't remember which one), I think it was 1+ protein. Not an issue he said and gave his reason.
That's key.
If they look comfortable, there is no medical emergency.
This is what the rabbit is missing.
"I felt weiirrd, so I took my blood pressure AND..." (while they're nimbly typing on their phone and eating garbage and fighting with their spouse).
I’m not sufficiently familiar with the test characteristics of a UA in ruling out an AKI in hypertensive emergencies to use it as a standalone test. That is to say, there is probably a reason why both ACEP and AHA recommend direct measurements of creatinine over a UA in that exceedingly uncommon scenario that you originally proposed.The point @VA Hopeful Dr was making was that we can easily do dipstick UAs in PCP clinic.
The whole point of my question, and subsequent posts, was how to keep patients OUT of the ED while doing the necessary things to evaluate for undifferentiated badness that might require an admission.
On the other hand, I do have practical reservations with using a UA over serum creatinine to accomplish your task. First, ED nurses seem extraordinarily afraid of piss. They literally seem to avoid eye contact with it. Typically, that amply filled cup will sit on the counter, filled to the rim with golden goodness, until discharge unless I hound the nurse to put a sticker on it and send it to the lab.
Second, I can’t get a clean catch urine to save my life. Inevitability, the sample is sent with a short-curly floater or a report of “motile sperm” from the patient’s earlier visit to Five Guys (apparently not just a burger joint). So, getting a “normal” UA that saves time on the back end (sorry, can’t help it) seems wishful.
Finally, there is the aspect of collegiality in the highly unusual situation that you proposed. If a PCP takes the time to contact me about their patient for whom they have a particular concern, I’m far more likely to send the requested test than perform an alternative in its stead.
So bottom line, if I’m worried about an AKI then I measure the renal function.
"I had this funny feeling in my pants"This is what the rabbit is missing.
"I felt weiirrd, so I took my blood pressure AND..." (while they're nimbly typing on their phone and eating garbage and fighting with their spouse).
"so I took my blood pressure"
👀
"When I had a funny feeling in my pants, my GF took care of that."
"I didn't think to check my BP."
"I had this funny feeling in my pants"
"so I took my blood pressure"
👀
"When I had a funny feeling in my pants, my GF took care of that."
"I didn't think to check my BP."
Like this is 90% of my asymptomatic HTN visits; they're not at all having a medical emergency, but they do have "first world problems."
I am a PCP and that post was in direct response to someone else asking what to do if you're in an outpatient office without a fast turn around time for labs.So there's blood and protein, now what? You do a Cr and it's 1.6 with nl lytes and no anion gap. So you... tell the patient to follow up with their PCP. Or maybe admit them to the special "Not a thing" ward where you pile the asymptomatic acute renal failures in with all the asymptomatic R/O DKA patients the clinic sends to us rather than managing their DM.
What do I do with ECG findings that show signs of hypertension, a BMP that's a little off, or... actually I don't even remember what I would want to see on fundoscope but I definitely wouldn't be able to see it anyway and don't care as an emergency physician for an asymptomatic chronic medical problem.That AHA paper says BMP to assess for end organ dysfunction (kidneys), EKG (heart), fundoscopic exam (retina).
So what you’re saying is don’t follow evidence & recs. Just enjoy the inevitable malpractice suit that will come their way.
Yup, makes sense.![]()
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Cool storyWhat do I do with ECG findings that show signs of hypertension, a BMP that's a little off, or... actually I don't even remember what I would want to see on fundoscope but I definitely wouldn't be able to see it anyway and don't care as an emergency physician for an asymptomatic chronic medical problem.
Understood, and a UA is not an unreasonable screen for kidney problems. But in the absence of concerning symptoms, I'd argue that you don't need a fast turnaround time on labs. I'd go even farther and say that approaching HTN as "number too high = emergency"actually sabotages the chronic management of HTN. Patients internalize fear over a very high number while also passively receiving the message that 170/90 must be ok because everyone didn't freak out at that number.I am a PCP and that post was in direct response to someone else asking what to do if you're in an outpatient office without a fast turn around time for labs.
To be clear, I'm not saying that you personally do this or are mismanaging your patients' blood pressure. This is just what I've seen from a representative sample of patients sent in to the ED for asymptomatic HTN over the last 20 yrs. And it's compounded by trends in hospital LOS and utilization management. In the day of yore, you could admit a non-compliant HTN patient and they would stay in the hospital until they were titrated to an appropriate, stable BP on oral meds. Now days, hospitals exist to stabilize patients enough that they will likely survive an outpatient course of treatment. And an asymptomatic patient is almost always, by definition, stable.
Preaching to the choir, but if you are a PCP who wants to have a reasonable way to prove that a patient's kidneys are not acutely failing this is a quick way to reasonably do so.Understood, and a UA is not an unreasonable screen for kidney problems. But in the absence of concerning symptoms, I'd argue that you don't need a fast turnaround time on labs. I'd go even farther and say that approaching HTN as "number too high = emergency"actually sabotages the chronic management of HTN. Patients internalize fear over a very high number while also passively receiving the message that 170/90 must be ok because everyone didn't freak out at that number.
To be clear, I'm not saying that you personally do this or are mismanaging your patients' blood pressure. This is just what I've seen from a representative sample of patients sent in to the ED for asymptomatic HTN over the last 20 yrs. And it's compounded by trends in hospital LOS and utilization management. In the day of yore, you could admit a non-compliant HTN patient and they would stay in the hospital until they were titrated to an appropriate, stable BP on oral meds. Now days, hospitals exist to stabilize patients enough that they will likely survive an outpatient course of treatment. And an asymptomatic patient is almost always, by definition, stable.
I don't do this anymore, but when I first got out of residency this is what I would do.
I’ve learned that these patients need everything yet also nothing. I think it’s reasonable to check an EKG and BMP in the ED. I also think it’s reasonable to do nothing and give them information for a new PCP.
I would not check an ECG in an asymptomatic patient with HTN - no matter the number. This will only lead to diagnostic confusion and more testing. That is because the most common abnormalities in HTN, LVH and strain patterns, are not indicative of acute end organ damage.
So... what's symptomatic hypertension? Please don't tell me headache, dizziness, lightheadedness, fatigue, etc. And no, ACS, AHDF, CVA and AKI don't count. Those are end organ damage and diagnoses. What symptom do you get from plain old hypertension? I hate our current nomenclature.
In before the rabbit dismisses you with "cool story, thanks".
Does anyone else work in a dept where everything above is the anomaly?
in my dept, these patient seem to always get a full panel of labs, including serial hsTrops, +/- cxr, +/- head CT, and generally a dose or two of po clonidine, IV labetalol or IV hydralazine. Many of these patients have an Rx for prn home clonidine for sbp > 180.
This often leads to repeat visits the next day b/c their bp is “back up” where I have to figure out a way to tactfully explain that my partners are either idiots or doing a bunch of unnecessary things in order to placate them.
in my dept, these patient seem to always get a full panel of labs, including serial hsTrops, +/- cxr, +/- head CT, and generally a dose or two of po clonidine, IV labetalol or IV hydralazine. Many of these patients have an Rx for prn home clonidine for sbp > 180.
This often leads to repeat visits the next day b/c their bp is “back up” where I have to figure out a way to tactfully explain that my partners are either idiots or doing a bunch of unnecessary things in order to placate them.
The diagnostic shotgun approach started getting out of control back when we began putting providers in triage (roughly 2010ish). This parallels the massive escalation in diagnostics that occurred with other conditions presenting to triage during that era.Does anyone else work in a dept where everything above is the anomaly?
in my dept, these patient seem to always get a full panel of labs, including serial hsTrops, +/- cxr, +/- head CT, and generally a dose or two of po clonidine, IV labetalol or IV hydralazine. Many of these patients have an Rx for prn home clonidine for sbp > 180.
This often leads to repeat visits the next day b/c their bp is “back up” where I have to figure out a way to tactfully explain that my partners are either idiots or doing a bunch of unnecessary things in order to placate them.
On the other hand, I’d say that aggressively treating these numbers has actually pulled back a bit over the past 15-20 years as more of us have come to the understanding that lowering a number without end organ damage is more harmful than beneficial.
What the AHA paper had hoped to accomplish was putting a final nail in the coffin of acutely managing elevated numbers that are not associated with end organ damage. There are still some holdouts largely driven by ancient nursing culture that force our hands. Case in point, we’ve all been told that the floor nurses refuse signout on a patient unless you get the BP below some arbitrary number. This AHA policy paper is finally an authoritative organization calling out such behavior as bad medicine.
My last shift was seeing 6 out of 8 of my first patients within the first 1.5 hrs of my shift as nothing but primary care complaints or PCP sent them. I didn’t care much because I was paid $400 per hr to see them. Took me 5 mins to see each one, 2 mins to write the note and 2 minutes to say good-bye.
My last shift was seeing 6 out of 8 of my first patients within the first 1.5 hrs of my shift as nothing but primary care complaints or PCP sent them. I didn’t care much because I was paid $400 per hr to see them. Took me 5 mins to see each one, 2 mins to write the note and 2 minutes to say good-bye.
What did you spend the other minute doing?
What do you do with the ECG when the patient feels fineI’ve learned that these patients need everything yet also nothing. I think it’s reasonable to check an EKG and BMP in the ED. I also think it’s reasonable to do nothing and give them information for a new PCP.
If I catch these orders from triage in time, I cancel them.Does anyone else work in a dept where everything above is the anomaly?
in my dept, these patient seem to always get a full panel of labs, including serial hsTrops, +/- cxr, +/- head CT, and generally a dose or two of po clonidine, IV labetalol or IV hydralazine. Many of these patients have an Rx for prn home clonidine for sbp > 180.
This often leads to repeat visits the next day b/c their bp is “back up” where I have to figure out a way to tactfully explain that my partners are either idiots or doing a bunch of unnecessary things in order to placate them.
LVH with left axis deviation and poor R wave progression is America's EKG. Neither reassuring nor diagnostic, it is the weak and dizzy of EKGs.I would not check an ECG in an asymptomatic patient with HTN - no matter the number. This will only lead to diagnostic confusion and more testing. That is because the most common abnormalities in HTN, LVH and strain patterns, are not indicative of acute end organ damage.
LVH with left axis deviation and poor R wave progression is America's EKG. Neither reassuring nor diagnostic, it is the weak and dizzy of EKGs.
Genius comment.
Chef's kiss.
I didn’t say I routinely do a workup on these patients but I understand the docs who do.What do you do with the ECG when the patient feels fine
But, I will say it’s nice to have a baseline EKG on some of these people because they’ll 100% be back at some point.
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btw in february or march of this year ACEP changed the recommendations in a really subtle way. They previously used to say that you didn't need to start any new meds, just pass it off the PCP (and if you read the FULL report a lot of that is that since blood work isnt necessary on these patients but blood work IS needed to start any med except ccbs, just dont start meds so you dont have to draw blood). Now they say that we *should* start meds and/or increase current dosages if the reason is not noncompliance or just pain/anxiousness. They still make it very clear that there is no goal BP or any "dangerous" BP to act differently over. They are just making day zero of new treatment be the ED visit.
I have no problem with the recommendations.
I have a problem with them taking down (and completely wiping the internet of) their old recommendations which had SUCH a good patient-friendly summary of the "dont test, don't treat, don't worry' mantra. Now they only have the overly-dense medical version of the new guidelines.
I have no problem with the recommendations.
I have a problem with them taking down (and completely wiping the internet of) their old recommendations which had SUCH a good patient-friendly summary of the "dont test, don't treat, don't worry' mantra. Now they only have the overly-dense medical version of the new guidelines.
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I'm sure everyone else has said it, but you can't educate the PCPs. Not that its impossible but that 51% of those referrals are actually coming from their triage nurse, waiting room receptionist, or answering service, not the PCP. 44% are coming from physicians who "have been practicing this way for years and nothing bad has happened yet" and are closed to the idea of changing their patterns. About 5% actually sent them over for some other reason but the patient is too fixated on the BP to even know why they got sent.You guys actually bother trying to educate other doctors on why it's pointless to send them in?
One of our PCP's sent in a woman who was ~200/100. No symptoms but apparently there were EKG changes. I looked at them and they were subtle STD or even non-existent. One of those kinds that you can barely see. I tried letting the PCP know there isn't much for us to do, and even said our national guidelines write to not treat and to let outpatient doctors treat. She was an outpatient doctor and sent her in anyway.
I don't even know if it's worth it these days.
btw in february or march of this year ACEP changed the recommendations in a really subtle way. They previously used to say that you didn't need to start any new meds, just pass it off the PCP (and if you read the FULL report a lot of that is that since blood work isnt necessary on these patients but blood work IS needed to start any med except ccbs, just dont start meds so you dont have to draw blood). Now they say that we *should* start meds and/or increase current dosages if the reason is not noncompliance or just pain/anxiousness. They still make it very clear that there is no goal BP or any "dangerous" BP to act differently over. They are just making day zero of new treatment be the ED visit.
I have no problem with the recommendations.
I have a problem with them taking down (and completely wiping the internet of) their old recommendations which had SUCH a good patient-friendly summary of the "dont test, don't treat, don't worry' mantra. Now they only have the overly-dense medical version of the new guidelines.
You referring to this?
Yup. The old page where they had a similar formal paper AND a simplified "two questions you need to know" based on the 2013 recommendations is gone. Which I understand based on them having new recommendations. But I really wish they left the old page because that "two questions you need to know" would need only the slightest modification to still be accurate and was so useful for reassuring patients that their asymptomatic htn is fine.
I think we do these people a disservice when we don't start antihypertensives. Most people can't see a PCP for an initial visit for at least six months.