HI
here there is some biochem qustions
lets do it and think about it
1. Birds require arginine in their diet. Would you expect to find the production of urea in these animals? Why
2. Why is glutamate dehydrogenase a logical point for the control of ammonia production in cells
3. After an overnight fast, muscle tissue produces amino acids like alanine, glutamate and glutamine. They are rapidly taken up by liver. What happens to these amino acids?
4. A male infant of six weeks was presented with symptoms of hyperammoneamia. He had low blood urea and elevated serum transaminases with aminoaciduria and hyperuricacideamia. The level of citrullin argininosuccinic acid and argininosuccinate was low. Enzyme assay established that CPSI is only 20% active which was fully active in the presence of NAG. As part of therapy supplement of arginine, pyridoxine and keto acids analogues of essential amino acids. Dietary protein was restricted.
A. Why were levels of blood ammonia high?
B. Explain why infants with CPSI deficiency normally exhibit relatively low blood levels of citrullin argininosuccinate arginine and urea.
C. Why is arginine prescribed as a supplement?
D. Why are keto acid and pyridoxine administers
E. Why was dietary protein restricted
F. Why would you expected to see elevated levels of glutamate, glutamine and alanine in the blood and urine of this patient
and i have many others i may post them later
all the best👍
here there is some biochem qustions
lets do it and think about it
1. Birds require arginine in their diet. Would you expect to find the production of urea in these animals? Why
2. Why is glutamate dehydrogenase a logical point for the control of ammonia production in cells
3. After an overnight fast, muscle tissue produces amino acids like alanine, glutamate and glutamine. They are rapidly taken up by liver. What happens to these amino acids?
4. A male infant of six weeks was presented with symptoms of hyperammoneamia. He had low blood urea and elevated serum transaminases with aminoaciduria and hyperuricacideamia. The level of citrullin argininosuccinic acid and argininosuccinate was low. Enzyme assay established that CPSI is only 20% active which was fully active in the presence of NAG. As part of therapy supplement of arginine, pyridoxine and keto acids analogues of essential amino acids. Dietary protein was restricted.
A. Why were levels of blood ammonia high?
B. Explain why infants with CPSI deficiency normally exhibit relatively low blood levels of citrullin argininosuccinate arginine and urea.
C. Why is arginine prescribed as a supplement?
D. Why are keto acid and pyridoxine administers
E. Why was dietary protein restricted
F. Why would you expected to see elevated levels of glutamate, glutamine and alanine in the blood and urine of this patient
and i have many others i may post them later
all the best👍
