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I am on a dermatology rotation at the moment, dealt with a late 20's male caucasian from northern Colorado with a chronic, spreading apparent fungal infection with unusually fast growth rate and visible consumption of organic material within minutes. I have not seen any infectious material consume organic material on such a varying and expediant basis, nor come in such a regular shape macroscopically whether grown in flesh, in sputum/intranasal sinus debris, or in fabric. Yes, it spreads from host to fabric and back to host. Suspect destruction of all clothing and bedding will be required, including synthetic as it will stick to and grow in synthetic fabric. I confirmed this after requesting clothing samples.
I do not have a picture of the microscopy or culture though it clearly has formative andcyeastlike characteristics. It will grow in most or dry areas but not wet. In the buttocks, it appears to grow out like, best possible description, like thick cornflakes at end of hair or, in crease, like bread crust. Conidia and buds present but loose in sample microscopy. Appears like tinea versicolor but is spreading and contaminating too widespread. Ketaconazole cream 2% ineffevtive after terbinifine 1% (Lamisil AT) was tried.
Macroscopic under skin appears as the or more red dots. Forms pustules which will spread in flavic manner, but appears to heal. The skin from healing is not skin however but skin consumed by the fungi or algae and quickly spread by suspension to cover open sores or uncovered organisms. The organisms macroscopically appear in a few forms:
1: clustered spheroids with white debris. Will often be accompanied by "cresent" on one side with a turret-like growing stem with a leaf-like tip that acts like a syringe. Will quickly reach for nearby organic matter and inflate with air/spore mixture.
2: Ascomycotic large scale organism which will have cone-shaped appendages taming in organic threads at visibly fast rate. Organism grabbed with threaded paper towel latched on and grew from about 1cmsq to 120% original size in 5 minutes. Appears to have comes for intake as well as one to three spore emitting nostrils and a large mouth-like opening next to. Smaller organisms only have one spore release but will have mouthlike opening, and are tubelike. These have been found deeply embedded cutaneously and subcutaneously in at least two existing places. Granulomae form immediately around thick cell walls as they form on these organisms, as to form a webbing of fungal and dermal material in fingertips, for example. Loss of sensation in these areas noted as a layer of suction cuplike film was found under epiderm.
3: cottony webs: the smallest form seen and commonly found in clothing are white or yellow cottony tangled webs with black or grey bulbs or material at center. At least two examples of the Type 2 formations found were also covered in these yellow microtubes.
Progression: patient watched as infection started in form of the cease in his buttocks being itchy and more most than usual. Instinctively scratching spread the infection to his fingers. A week after estimated start he noticed yellow "hairs" sprouting from his fingers. He figured it was normal until one such fiber launch from one finger and penetrated another, stinging in the process. This allegedly continued almost constantly. Fungal formations under nails formed, like thick fibrous arms extending outward. He then found small pustules on hands and forearms. These were picked open revealing offgreen yellow fluid with a cluster as described in form A. He continued to pick until his arms were coveted in favic sores. These "would quickly cover, spreading to heal in a day or less almost completely until adopting lamisil." Lamisil uncovered layers of material on sores but did not cute the infection, only slowed it. Jis physician prescribed ketaconazole creme 2% and diagnosed it tinea versicolor. The ketaconaloze was barely more effective than lamisil but did not cure any of the infected areas. Parts of his finger tips opened up with granulomic blisters. Dead fungus left, but as he picked areas should visibly grow in short time more fungal material. The liquid from the sores dried in offwhite or brown debris clusters. Infection spread to IN Sinus, naturally from infection of buttocks. Patient coughs up and sneezes out light green material in dame forms. "Algae bulbs" appear mixed in with fungal material.
The organism will consume organic material attached to rigid inorganic material at such a rate and intensity that it will erode aluminum foil (see image). It all also latch and consume warm foods, specifically those with oil or protein and carbohydrates, at a very fast and visible rate and is detectable by texture.
Anyone know what this might be?
I do not have a picture of the microscopy or culture though it clearly has formative andcyeastlike characteristics. It will grow in most or dry areas but not wet. In the buttocks, it appears to grow out like, best possible description, like thick cornflakes at end of hair or, in crease, like bread crust. Conidia and buds present but loose in sample microscopy. Appears like tinea versicolor but is spreading and contaminating too widespread. Ketaconazole cream 2% ineffevtive after terbinifine 1% (Lamisil AT) was tried.
Macroscopic under skin appears as the or more red dots. Forms pustules which will spread in flavic manner, but appears to heal. The skin from healing is not skin however but skin consumed by the fungi or algae and quickly spread by suspension to cover open sores or uncovered organisms. The organisms macroscopically appear in a few forms:
1: clustered spheroids with white debris. Will often be accompanied by "cresent" on one side with a turret-like growing stem with a leaf-like tip that acts like a syringe. Will quickly reach for nearby organic matter and inflate with air/spore mixture.
2: Ascomycotic large scale organism which will have cone-shaped appendages taming in organic threads at visibly fast rate. Organism grabbed with threaded paper towel latched on and grew from about 1cmsq to 120% original size in 5 minutes. Appears to have comes for intake as well as one to three spore emitting nostrils and a large mouth-like opening next to. Smaller organisms only have one spore release but will have mouthlike opening, and are tubelike. These have been found deeply embedded cutaneously and subcutaneously in at least two existing places. Granulomae form immediately around thick cell walls as they form on these organisms, as to form a webbing of fungal and dermal material in fingertips, for example. Loss of sensation in these areas noted as a layer of suction cuplike film was found under epiderm.
3: cottony webs: the smallest form seen and commonly found in clothing are white or yellow cottony tangled webs with black or grey bulbs or material at center. At least two examples of the Type 2 formations found were also covered in these yellow microtubes.
Progression: patient watched as infection started in form of the cease in his buttocks being itchy and more most than usual. Instinctively scratching spread the infection to his fingers. A week after estimated start he noticed yellow "hairs" sprouting from his fingers. He figured it was normal until one such fiber launch from one finger and penetrated another, stinging in the process. This allegedly continued almost constantly. Fungal formations under nails formed, like thick fibrous arms extending outward. He then found small pustules on hands and forearms. These were picked open revealing offgreen yellow fluid with a cluster as described in form A. He continued to pick until his arms were coveted in favic sores. These "would quickly cover, spreading to heal in a day or less almost completely until adopting lamisil." Lamisil uncovered layers of material on sores but did not cute the infection, only slowed it. Jis physician prescribed ketaconazole creme 2% and diagnosed it tinea versicolor. The ketaconaloze was barely more effective than lamisil but did not cure any of the infected areas. Parts of his finger tips opened up with granulomic blisters. Dead fungus left, but as he picked areas should visibly grow in short time more fungal material. The liquid from the sores dried in offwhite or brown debris clusters. Infection spread to IN Sinus, naturally from infection of buttocks. Patient coughs up and sneezes out light green material in dame forms. "Algae bulbs" appear mixed in with fungal material.
The organism will consume organic material attached to rigid inorganic material at such a rate and intensity that it will erode aluminum foil (see image). It all also latch and consume warm foods, specifically those with oil or protein and carbohydrates, at a very fast and visible rate and is detectable by texture.
Anyone know what this might be?
