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Okay, in the midst of studying for my orals I came across several questions that I would like to get some professional opinions on:
1) Can you place an epidural in a patient with normal pressure hydrocephalus? (obviously you can but what if you wet tap them)
2) When you release the aortic cross clamp for a TAA repair is the increase in end tidal CO2 due to the increase in the pooled acidic (high CO2) blood to the lungs? (or is it is due to increased production or decrease in deadspace--these 2 concepts were brought up in Ho's practice exams and I wasn't sure. I didn't think increased production or deadspace had anything to do with the rise but now I am not sure).
1) Can you place an epidural in a patient with normal pressure hydrocephalus? (obviously you can but what if you wet tap them)
2) When you release the aortic cross clamp for a TAA repair is the increase in end tidal CO2 due to the increase in the pooled acidic (high CO2) blood to the lungs? (or is it is due to increased production or decrease in deadspace--these 2 concepts were brought up in Ho's practice exams and I wasn't sure. I didn't think increased production or deadspace had anything to do with the rise but now I am not sure).