Epinephrine/Norepinephrine/Cortisol and a question about the thalamus
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Cortisol is a glucocorticoid (and steroid hormone) released from the adrenal cortex in response to stress or low glucose levels. ACTH from the anterior pituitary stimulates its release. Cortisol will increase blood sugar levels through gluconeogenesis (making of glucose using non-carbohydrate sources -- this occurs mainly in the liver).
Epinephrine/Nor are tyrosine derivatives released from the adrenal medulla. They act through the sympathetic nervous system, commonly known as "fight or flight response." They increase heart rate and blood glucose levels through glycogenesis (conversion of glycogen to glucose in the liver) The conversion of glycogen to glucose is also what glucagon from the pancreas accomplishes.
Think gluconeogenesis = neo = new sources; glycogenesis = glyco = glycogen
Please correct me if I am wrong, anyone
To clarify admackbar a bit glyocogen stores are mainly in the liver (muscles to), and the liver is where the glucogenesis happens (not the pancreas). I can see how you are confusing cortisol and norepi/epi but coritsol is more of a long term response to stress, with epi being a immediate response to extreme stress. Of course there effects are also completely different. It may also be worth knowing there are several receptor subtypes for epinephrine, so it does not do the same thing in each part of the body.
To answer the other part of your question the thalmus is the "motherboard" and not the hypothalmus.
To answer the other part of your question the thalmus is the "motherboard" and not the hypothalmus.
