Euvolemic hyponatremia and water retention

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Daitong

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So SIADH or any causes of pathologically increased ADH results in euvolemic hyponatremia, but I'm confused to how the body stays euvolemic- there is more water reabsorbed, and no edema develops, and there is no hypertension (no additional vascular blood), so where does the reabsorbed water go?

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Excessive ADH secretion in SIADH leads to water retention, which leads to transient extracellular fluid expansion. This decreases aldosterone and increases natriuretic peptides causing the loss of sodium through urine, so this way the extra cellular fluid volume normalizes.
 
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