B Bandwagon1 Full Member 10+ Year Member Oct 2, 2015 #1 Advertisement - Members don't see this ad In Hyperchylomicronemia, if apoC II deficiency or the LPL defect causes this disease how come we only see an increase in chylomicrons but not VLDLs? I thought that VLDLs also needs apoC to be cleaved by LPL.
Advertisement - Members don't see this ad In Hyperchylomicronemia, if apoC II deficiency or the LPL defect causes this disease how come we only see an increase in chylomicrons but not VLDLs? I thought that VLDLs also needs apoC to be cleaved by LPL.
