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Kaplan explains this it's because the liver starts throwing out lipid to compensate the loss of protein due to nephrotic syndrome. To me this doesn't really make any sense. Lipid breakdown via beta-oxidation will generate ATP that can be used for gluconeogenesis, but w/o glucogenic amino acids, gluconeogenesis won't work. So what is the point of releasing lipid into the blood stream if you are losing your protein in nephrotic syndrome?
Can anyone explain the hyperlipidemia in nephrotic syndrome?
Many thanks in advance.
Can anyone explain the hyperlipidemia in nephrotic syndrome?
Many thanks in advance.