Can anyone tell me why we see Hypokalemia a/c with Alkalosis; and Hyperkalemia with Acidosis?
I know that because in Alkalosis, the blood PH is too high, so inorder to counter that, the intracellullar H ions go out of the cells into the blood, using the H-K pump, pushing K into the cells, and as such decreasing the serum K levels (vice versa in Acidosis)
But I'm asking this from a renal point of view. Let's say in Heart Failure, the Renin-Angiontensin system becomes active, increases Aldosterone, which retains Sodium and excretes K and body becomes hypokalemic. In this scenario, where does the Alkalosis come from?
I know that because in Alkalosis, the blood PH is too high, so inorder to counter that, the intracellullar H ions go out of the cells into the blood, using the H-K pump, pushing K into the cells, and as such decreasing the serum K levels (vice versa in Acidosis)
But I'm asking this from a renal point of view. Let's say in Heart Failure, the Renin-Angiontensin system becomes active, increases Aldosterone, which retains Sodium and excretes K and body becomes hypokalemic. In this scenario, where does the Alkalosis come from?