How can we explain the presence of hyponatriuria in patient with cronich heart failure since, in this case, there usually is hyponatriemia? The question is: how can these two be compatible with each other? I know there is an RAA and ADH activation but I used to think the ADH system activation was more powerfull and this led to hyponatriemia. Unluckily, this collide with the finding of hyponatriuria. What am I missing?