In mitral stenosis is there dilation, hypertrophy or both?

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cooldude89

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In mitral stenosis is there dilation, hypertrophy or both?

Dilation bec of blood coming from lungs into atria increase, and in first aid it says mitral stenosis lead to enlargement left atrium in MS causing dilation right?

Hypertrophy bec the atria needs to push against the stenotic valve to get blood across to left ventricle ...right

But I thought you could not get the phenomenon of hypertrophy and dilation together??
 
I believe it's both dilation and hypertrophy because of the reasons you've already mentioned.. They could happen together.
 
In mitral stenosis is there dilation, hypertrophy or both?

Logically, dilation.

Dilation bec of blood coming from lungs into atria increase, and in first aid it says mitral stenosis lead to enlargement left atrium in MS causing dilation right?

Why would blood coming from lungs into atrium increase? Blood going out of atrium, on the other hand, does decrease.

Hypertrophy bec the atria needs to push against the stenotic valve to get blood across to left ventricle ...right

What is going to ask the left atrium to push harder?

But I thought you could not get the phenomenon of hypertrophy and dilation together??

Who says so? Quite often one leads to the other, and often, both coexist.
 
Mitral stenosis-> increased pressure in LA->dilation and hypertrophy of LA ( p waves of LA hypertrophy in D II, D III and V1)-> backup of blood in the pulmonary veins-> venous pulm. hypertension-> arterial pulmonary hypertension-> hypertrophy of RV-> dilation and failure of RV-> right heart failure signs (JVD, hepatomegaly, ascites, peripheral edema).
LV is normal/ with decreased dimensions if no other valvulopathies are present.
End-stage of pulm.hypertension=parenchimal modifications (insterstital edema, alveolar edema, pulmonary hemosiderosis)
 
Mitral stenosis-> increased pressure in LA->dilation and hypertrophy of LA ( p waves of LA hypertrophy in D II, D III and V1)-> backup of blood in the pulmonary veins-> venous pulm. hypertension-> arterial pulmonary hypertension-> hypertrophy of RV-> dilation and failure of RV-> right heart failure signs (JVD, hepatomegaly, ascites, peripheral edema).
LV is normal/ with decreased dimensions if no other valvulopathies are present.
End-stage of pulm.hypertension=parenchimal modifications (insterstital edema, alveolar edema, pulmonary hemosiderosis)

this guys answer is complete with EKG findings and everything. I like it!
 
Mitral stenosis-> increased pressure in LA->dilation and hypertrophy of LA ( p waves of LA hypertrophy in D II, D III and V1)-> backup of blood in the pulmonary veins-> venous pulm. hypertension-> arterial pulmonary hypertension-> hypertrophy of RV-> dilation and failure of RV-> right heart failure signs (JVD, hepatomegaly, ascites, peripheral edema).
LV is normal/ with decreased dimensions if no other valvulopathies are present.
End-stage of pulm.hypertension=parenchimal modifications (insterstital edema, alveolar edema, pulmonary hemosiderosis)

Could you explain why there will be LA hypertrophy in MS? What is the physiopathology/mechanism behind that?
 
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