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- Jan 31, 2008
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Hello everybody. MS4 with questions about Laryngospasm.
1. First has to do with basic recognition and differential diagnosis of stridor following extubation. A review article I was reading states "Both supraglottic obstruction and partial laryngospasm are associated with inspiratory stridor and intercostal retractions with rapidly deteriorating oxygenation." What are the causes of supraglottic obstruction that I should be aware of for differential purposes. Supraglottic obstruction just sounds vague to me and I'm not sure what exactly causes it. What else should be on my differential that may mimic partial or complete laryngospasm?
2. The same review article discusses the use of propofol before resorting to sux in a linear algorithm after jaw thrust, CPAP, pulling of mandible, and pressure in a special notch fail. Is this commonly practiced? (it cites 75% percent of the time propofol will releive the spasm). It would make me nervous in a desaturating patient wait for some period of time to see if this is going to be that one out of four cases where propofol isn't going to help. Does your choice depend on the O2 sat? I also saw a two armed algorithm based on complete vs. partial obstruction. Would anybody be willing to share pearls on how they practice or organize the treatment algorithms in their mind?
1. First has to do with basic recognition and differential diagnosis of stridor following extubation. A review article I was reading states "Both supraglottic obstruction and partial laryngospasm are associated with inspiratory stridor and intercostal retractions with rapidly deteriorating oxygenation." What are the causes of supraglottic obstruction that I should be aware of for differential purposes. Supraglottic obstruction just sounds vague to me and I'm not sure what exactly causes it. What else should be on my differential that may mimic partial or complete laryngospasm?
2. The same review article discusses the use of propofol before resorting to sux in a linear algorithm after jaw thrust, CPAP, pulling of mandible, and pressure in a special notch fail. Is this commonly practiced? (it cites 75% percent of the time propofol will releive the spasm). It would make me nervous in a desaturating patient wait for some period of time to see if this is going to be that one out of four cases where propofol isn't going to help. Does your choice depend on the O2 sat? I also saw a two armed algorithm based on complete vs. partial obstruction. Would anybody be willing to share pearls on how they practice or organize the treatment algorithms in their mind?