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- May 9, 2013
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Can I list this as a hobby in my activities section? Any tips on how to (and how not to) present it? This is one of my strongest motivations for going into medicine and it's why I'm such a proponent of evidence-based medicine. However, I'll drop it if it's a red flag. I don't have much formal research experience; I just started volunteering in a circadian lab.
As a hobby, I've been reading scientific papers since 2012 and collecting and annotating them since 2015. I read about a wide variety of topics, but most of my reading centers on underlying mechanisms that drive the pathogenesis of many chronic diseases, especially mitochondrial dysfunction and chronic inflammation in neurodegenerative and metabolic disorders.
As an example, a paper I just started reading is
Obesity, diabetes mellitus, atherosclerosis and chronic periodontitis: a shared pathology via oxidative stress and mitochondrial dysfunction? I'm also very interested in promising approaches to reverse these pathologies for the purpose of slowing the progression of chronic diseases. For instance, Nrf2 inducers and membrane lipid replacement therapy to reverse acquired mitochondrial dysfunction to slow the progression of neurodegenerative and metabolic disorders.
For any skeptics in the audience, I'm happy to provide higher-quality evidence for these ideas than the reviews I've linked to.
As a hobby, I've been reading scientific papers since 2012 and collecting and annotating them since 2015. I read about a wide variety of topics, but most of my reading centers on underlying mechanisms that drive the pathogenesis of many chronic diseases, especially mitochondrial dysfunction and chronic inflammation in neurodegenerative and metabolic disorders.
As an example, a paper I just started reading is
Obesity, diabetes mellitus, atherosclerosis and chronic periodontitis: a shared pathology via oxidative stress and mitochondrial dysfunction? I'm also very interested in promising approaches to reverse these pathologies for the purpose of slowing the progression of chronic diseases. For instance, Nrf2 inducers and membrane lipid replacement therapy to reverse acquired mitochondrial dysfunction to slow the progression of neurodegenerative and metabolic disorders.
For any skeptics in the audience, I'm happy to provide higher-quality evidence for these ideas than the reviews I've linked to.