need help answering bio question!

[biostudent101]

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1. in addition to urogenital infections Nesserie gonorrhea is ocassionally also isolated from:
a. skin
b. pharynx
c. nasl cavity
d. blood
e. scalp

2. cacer cells can be inactivated by:
a. chemical agents that inactivated DNA polymerase
b. specific antibodies that could bin to the malignant cells
c. angiotensins I and II
d. interferons
e. all of the above

3.ebola viruses are transmitted by:
a. fecal- oral route
b. drinking water
c. insects bite
d. air borne
e. sexual contacts

4. a physiological response to hyponatremia in water movements:
a. out of the cells
b. into the cells
c. into the intertestial spaces of the cells
d. none of the above
e. all of the above
these are pathophysiology Questions
thanks in advance

 

[doc3232]

These won't be on the DAT

I think A B D for first 3.

 

[smurf528]

wow. I don't even know any of these. I hope this isn't how the actual DAT is going to be.

 

[doc3232]

wow. I don't even know any of these. I hope this isn't how the actual DAT is going to be.

It wouldn't be, but you should have an idea for the 2nd one.

 

[Contach]

It wouldn't be, but you should have an idea for the 2nd one.

im curious to know why you picked B for the second question:
2. cacer cells can be inactivated by:
a. chemical agents that inactivated DNA polymerase
b. specific antibodies that could bin to the malignant cells
c. angiotensins I and II
d. interferons
e. all of the above

one of the biggest challenges to fighting cancer is identifying the cancer cells. The thing is, the cancer cells are just like all the other cells in your body, except maybe they will lack an inhibition factor or have an excess growth inducer protein. But these things are within the cell and rarely shown on the surface. If the cancer cells did show specific antigens or cell surface markers, your immune response would easily be able to identify it and opsonize/phagoctyose/kill it. Also, if cancer had specific cell surface markers on the outside of the cells, it would make fighting cancer much easier than it currently is: give a dose of antibodies and BAM the cancer is contained. It is relatively easy to make antibodies.

On the other hand you should know that DNA polymerase is involved in DNA synthesis and thus needed for mitosis (increasing the number of cells - hallmark of cancer). Thus if you stopped DNA polymerase and hence the cell cycle's "S" stage you could inactivate a cancerous cell from dividing again and thus inactivating a cancerous cell. While there are also problems with doing this it is a more sound technique than "specific antibodies that bind to the cancerous cell surface." The cell cycle is where the genetic modificaitons of cancerous cells occur. The p52 (i think) protein is a growth regulator, and it is a lack of this protein that is responsible for pushing the cell to keep dividing. This means that p52 is responsible for inhibiting the progress of the cell cycle.

Thus I think the answer is B, and I barely even take science courses.

 

[Contach]

4. a physiological response to hyponatremia in water movements:
a. out of the cells
b. into the cells
c. into the intertestial spaces of the cells
d. none of the above
e. all of the above

something is wrong with the wording of this question, I hope you picked up on this. As written, it makes little sense..

I think you should know what hyponatremia is though...

 

[911dds]

what's the answer? If these kinds of questions are on the DAT...umm...I might as well give up right now.

 

[doc3232]

im curious to know why you picked B for the second question:
2. cacer cells can be inactivated by:
a. chemical agents that inactivated DNA polymerase
b. specific antibodies that could bin to the malignant cells
c. angiotensins I and II
d. interferons
e. all of the above

one of the biggest challenges to fighting cancer is identifying the cancer cells. The thing is, the cancer cells are just like all the other cells in your body, except maybe they will lack an inhibition factor or have an excess growth inducer protein. But these things are within the cell and rarely shown on the surface. If the cancer cells did show specific antigens or cell surface markers, your immune response would easily be able to identify it and opsonize/phagoctyose/kill it. Also, if cancer had specific cell surface markers on the outside of the cells, it would make fighting cancer much easier than it currently is: give a dose of antibodies and BAM the cancer is contained. It is relatively easy to make antibodies.

On the other hand you should know that DNA polymerase is involved in DNA synthesis and thus needed for mitosis (increasing the number of cells - hallmark of cancer). Thus if you stopped DNA polymerase and hence the cell cycle's "S" stage you could inactivate a cancerous cell from dividing again and thus inactivating a cancerous cell. While there are also problems with doing this it is a more sound technique than "specific antibodies that bind to the cancerous cell surface." The cell cycle is where the genetic modificaitons of cancerous cells occur. The p52 (i think) protein is a growth regulator, and it is a lack of this protein that is responsible for pushing the cell to keep dividing. This means that p52 is responsible for inhibiting the progress of the cell cycle.

Thus I think the answer is B, and I barely even take science courses.

Hmm...so I barely even read answer choice B...sort of just eliminated the others. I kicked A off for the same reason you eliminated it.
I remember reading something about cancer cells actually having receptors of some type. I think I read it in Cliffs.