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- Jul 11, 2001
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Greetings, 🙂
I'm confused about a particular topic: rib raising. The way I understood the technique was that while it initially increases sympathetic tone, after a while, sympathetic tone is decreased, and the goal is to decrease sympathetic tone and increase parasympathetic tone.
So my question sometime last year was, why would you do rib raising in asthma?. . . the sympathetic nervous system is responsible for bronchoconstriction.
I had asked this of a professor, and then I had also came across this from a web site:
"Lung adrenergic receptors are minimally innervated by sympathetic fibres but may be stimulated by circulating catecholamines or specific adrenergic agonists. "
So combining what the professor said and the above information, it all made sense to me: you decrease the sympathetic nervous imput and it reduces the amount of vasoconstriction and makes the secretions less viscous (because parasympathetic is vasodilation and less viscous secretions), and you have no impact on the degree of bronchoconstriction because those receptors are not innervated by specific nerves that come from T1-L2.
But then in class the other day, another professor stated that the reason rib raising works in asthma is by increasing sympathetic tone.
So I was wholly confused.
I tried to find the above quote again, and I did, only it was on the following website:
http://education.vetmed.vt.edu/Curriculum/local/VMS6534/respir/rb/autonomicreview.doc
which means it applies to horses. It could also apply to human lungs, but I found no evidence either way.
Could someone help me resolve this dilemma? Are lung B receptors directly innervated or not?
I'm confused about a particular topic: rib raising. The way I understood the technique was that while it initially increases sympathetic tone, after a while, sympathetic tone is decreased, and the goal is to decrease sympathetic tone and increase parasympathetic tone.
So my question sometime last year was, why would you do rib raising in asthma?. . . the sympathetic nervous system is responsible for bronchoconstriction.
I had asked this of a professor, and then I had also came across this from a web site:
"Lung adrenergic receptors are minimally innervated by sympathetic fibres but may be stimulated by circulating catecholamines or specific adrenergic agonists. "
So combining what the professor said and the above information, it all made sense to me: you decrease the sympathetic nervous imput and it reduces the amount of vasoconstriction and makes the secretions less viscous (because parasympathetic is vasodilation and less viscous secretions), and you have no impact on the degree of bronchoconstriction because those receptors are not innervated by specific nerves that come from T1-L2.
But then in class the other day, another professor stated that the reason rib raising works in asthma is by increasing sympathetic tone.
So I was wholly confused.
I tried to find the above quote again, and I did, only it was on the following website:
http://education.vetmed.vt.edu/Curriculum/local/VMS6534/respir/rb/autonomicreview.doc
which means it applies to horses. It could also apply to human lungs, but I found no evidence either way.
Could someone help me resolve this dilemma? Are lung B receptors directly innervated or not?