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akg

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After an automobile accident , a patient's chin deviates to the left on opening mouth widely.His mandible is probably fractured at which of the following points?

a. genial tubercle
b.left condyolar neck
c.right condylar neck
d.left mental foramen
e.right mental foramen

post the correct anwer and justify it with right explanation
 
Which of the following muscles has an important influence on the function of the mandible, although it is not generally considered a muscle of mastication?

A. Omohyoid
B. Geniohyoid
C. Mylohyoid
D. Digastric
E. Sternocleidomastoid

Thanks in advance.

hi,
answer is digastric
 
anterir belly of digastric , mylohyoid and also geniohyoid are all accesory muscle of mastication so the renaining omohyoid and sternocledo mastoid have to be seen but both these muscle are no where related to mastication omohyoid pulls the hyoid down one of the infrahyoid muscle so realy confused yar .lots of difference given about musle of mastication in decs, kaplan chaurasia and google search so someone can pls make a conformed muscle of mastication and accesory muscle of mastication for the nbde exam
 
Hi,

To answer this Q we need to use the formula ciVi= cfVf
where c= concentration
V = volume
i= initial values
f= final values

Substituting values for the formula

600* 1 = Mf * 4 ( not 3 for 3litres of water was added to 1litre
of solution)
Solving, Mf = 600/4 = 150 ml/l

To convert milliliters to grams, multiply the number in ml with specific gravity of the substance. for glucose, it is 1.44 So 150 ml * 1.44 = 216 gms = 216,000mg

Now, 1mmol of glucose = 180mg of glucose
So, the final value of glucose in mmol after dilution is 216,000/180 = 1200mmol.

hey buddys, I had to do a lot of searching to finally get this answer. but does any one think that such complicated questions will be asked for the exam. anyway, correct me if I'm wrong,k.

that is indeed a great search. But dont you think the answer should be only150 ml/l because the question is in ml/l, not in milimoles
 
that is indeed a great search. But dont you think the answer should be only150 ml/l because the question is in ml/l, not in milimoles

As per the question, the answer should be in mm/l which is a short form for mmol or millimolar. But, it indeed is a weird question. I believe questions like this shouldn't be asked for the exam.
 
Can anyone help in these question
Which of the following is not true about Hepatitis A infection
1) High morbidity rate
2) High mortality rate (ANS)
3) May be transmitted via fecal matter
4) May be transmitted via blood products
Shouldnt the answer be D as Hepatitis B is tranmitted by blood products.......

The mechanism of fibrin formation in damaged tissue is initiated by
1) Release of thromboplastin from damaged cells (ans)
2) release of ca from damaged cells
3) formation of thrombin from intercation of prothrombin and fibrinogen
4) interaction of thrombin and calcium
Why can't the answer be D?
 
Can anyone help in these question
Which of the following is not true about Hepatitis A infection
1) High morbidity rate
2) High mortality rate (ANS)
3) May be transmitted via fecal matter
4) May be transmitted via blood products
Shouldnt the answer be D as Hepatitis B is tranmitted by blood products.......

HA is transmitted through GIT,others include Polio, Rota.so 3 excluded early recovery so 2 should be answer,early recovery so 1 excluded. but dont know much about 4 as HA can transmit through fecal matter

The mechanism of fibrin formation in damaged tissue is initiated by
1) Release of thromboplastin from damaged cells (ans)
2) release of ca from damaged cells
3) formation of thrombin from intercation of prothrombin and fibrinogen
4) interaction of thrombin and calcium
Why can't the answer be D?

prothrombin coverstion requires thromboplastin and ca.so 3,4 excluded .anybody can explain??
 
Hep A is also transmitted by blood , but rarely.......the choice is " may be transferred by blood" ...... so you cant rule that out.
The correct answer is choice D: Hepatitis A never has a high mortality rate, but it DOES have a high morbidity rate.

Hope this helps
 
Hi,
I'm confused by the decks as it says that elevated level of aa will promote release of both. Is it correct? I thought elevated level of aa only promotes release of insulin. And elevated level of aa is an effect of glucagon. Can someone please confirm?
Thanks.
 
Thanks for the link......... It shows that thromboplastin is secreted by damaged cells.
QUOTE=confused tooth;4140810]Thromboplastin causes chemotaxis and attraction of plateles which plug the vessel, and also realease thromboxane which causes vasoconstriction ..

http://faculty.ucc.edu/biology-potter/hemostasis.htm

Correct me if I am wrong here[/QUOTE]
 
Can anybody explain?
The pulmonary neoplasm to which the endocrine effect of hyperparathyroudism is attributed
1) adenocarcinoma
2) oat cell carcinoma (ans)
3) pheochromocytoma
4)medullary carcinoma
5) squamous cell carcinoma

In which of the following organs or tissues ascending infections common
1)Bone
2)Kidney(ans)
3) Heart
4) Peritonium
5) Subcutaneous connective tissue
What do you mean by ascending infections?????

To which of the following sites does carcinoma of prostate metatstatis with greatest frequency
1) lung
2)liver
3)brain
4) spleen
5) skeleton ( ans)
 
The pulmonary neoplasm to which the endocrine effect of hyperparathyroudism is attributed
1) adenocarcinoma
2) oat cell carcinoma (ans)
3) pheochromocytoma
4)medullary carcinoma
5) squamous cell carcinoma
oatcell is paraneoplastic
In which of the following organs or tissues ascending infections common
1)Bone
2)Kidney(ans)
3) Heart
4) Peritonium
5) Subcutaneous connective tissue
What do you mean by ascending infections?????
can anybody explain? is it telling about pyelonephritis??

To which of the following sites does carcinoma of prostate metatstatis with greatest frequency
1) lung
2)liver
3)brain
4) spleen
5) skeleton ( ans)[/QUOTE]

exception is prostate it spreads dirtectly to bone.somebody correct me if i am wrong
 
thanks swetha.
The pulmonary neoplasm to which the endocrine effect of hyperparathyroudism is attributed
1) adenocarcinoma
2) oat cell carcinoma (ans)
3) pheochromocytoma
4)medullary carcinoma
5) squamous cell carcinoma
oatcell is paraneoplastic
In which of the following organs or tissues ascending infections common
1)Bone
2)Kidney(ans)
3) Heart
4) Peritonium
5) Subcutaneous connective tissue
What do you mean by ascending infections?????
can anybody explain? is it telling about pyelonephritis??

To which of the following sites does carcinoma of prostate metatstatis with greatest frequency
1) lung
2)liver
3)brain
4) spleen
5) skeleton ( ans)

exception is prostate it spreads dirtectly to bone.somebody correct me if i am wrong[/QUOTE]
 
initially, the developing heart is

a) induced by the notochord
b)positioned posterior to the notochord
c)positioned anterior to the prochordal plate
d)between the prochordal plate and the notochord



can any one find me the right answer, pls
 
heart is derived from mesoderm ,it lies cephalic to bcopharyngeal membrane and nueralplate and later it is ventral to forgut and caudal to buccopharyngeal membrane .so the ans can be either c or d , i think d is more appropriate
pls do correct me if i am wrong
 
heart is derived from mesoderm ,it lies cephalic to bcopharyngeal membrane and nueralplate and later it is ventral to forgut and caudal to buccopharyngeal membrane .so the ans can be either c or d , i think d is more appropriate
pls do correct me if i am wrong



As far as i rememebr things from my classroom , its cardiogenic plate,

but which one is gonna match to cardiogenic plate , need to be figured out though
 
Can anyone explain
which of the following is not required for normal blood clotting response?
1) Ca
2) plasmin ( ans)
3) thrombin
4) Vit K
5) phospholipid
6) proteolysis

Which of the follwing leave the body by the way of lungs
1) lipids
2) acetone (ans)
3)glucose
4)chlorides
5) amini acids
6) all of the above
7) none
 
Can anyone explain
which of the following is not required for normal blood clotting response?
1) Ca
2) plasmin ( ans)
3) thrombin
4) Vit K
5) phospholipid
6) proteolysis

plasmin because plasminogen coverted to plasmin by tissue plasminogen activator which aids in fibrin dissolution.
Which of the follwing leave the body by the way of lungs
1) lipids
2) acetone (ans)
3)glucose
4)chlorides
5) amini acids
6) all of the above
7) none

remeber that acetone smell in diabetics but dont know much about it,can anyone explain??
 
i was surfing for acetone and I found this......
"It is well known that under conditions which lead to the
formation and accumulation of the "acetone bodies," acetone
is present in the breath. It occurred to one of us that this excretion
of acetone from the lungs might be the result merely of
evaporation from the blood plasma into the alveolar air, conditioned
by the distribution coefficient of acetone between plasma
and air at the body temperature.
The characteristic odor of the breath of patients showing symptoms of diaLetic coma and the fact that this odor is due atleast in part to acet'one. Although most of workers who have contributed
to this subject have devoted their attention to the excretion
of these substances by the kidneys but there have been many
observations on the excretion of acetone by the lungs.
 
great search ,to add even alcohol excretion is throgh lungs and kidney
i had a doubt about occlusion which i have alredy posted in another thread but got no ans from any one ,hope to get it atleast now .there is a dicripancy in kaplan and 2004 decs on oclusion of premolars one says it occuds in triangular fossa and the other says on marginal ridge which one is correct what does the new 2006 dec say, pllllllllllllls help
 
Acc to 2006 decks
occlussion occurs in fossa
For Mand premolars
Distal triangular fossa
Mand molars 1 molar MBcusp- MMR of 1 molar and DMR of 2nd premolar
DB- central fossa
distal cusp-distal fossa
Max premolars
Mesial triang fossa
Max molars 1st Molar ML- central fossa
DL -DMR of 1st molar and MMR of 2nd molar
I''ll go with traingular fossa as I saw the same in UOP lectures

great search ,to add even alcohol excretion is throgh lungs and kidney
i had a doubt about occlusion which i have alredy posted in another thread but got no ans from any one ,hope to get it atleast now .there is a dicripancy in kaplan and 2004 decs on oclusion of premolars one says it occuds in triangular fossa and the other says on marginal ridge which one is correct what does the new 2006 dec say, pllllllllllllls help
 
Well ,thanks For Conforming It .but What Is Uop Lecture And For Biochem Isnt The Decs And Kap Book Enough
 
I have few uop school dental anatomy lectures nothing . I just used them for reviewing. I think decs and kap book for bioch is enough.
Its not about how much more you know. its more about one having good grip of the subject.......
I might be wrong!!!!
 
Can anybody explain
Acetylcholine is released at all of the following junctions expect
1-sympthatic ganglia
2-parasympthatic ganglia
3-somataic efferts to skeletal muscles
4-terminal sympathetic fibers to heart (ans)
5-terminal sympathetic fibers to adrenal medulla
Why can't the ans be 5?
 
i was surfing for acetone and I found this......
"It is well known that under conditions which lead to the
formation and accumulation of the “acetone bodies,” acetone
is present in the breath. It occurred to one of us that this excretion
of acetone from the lungs might be the result merely of
evaporation from the blood plasma into the alveolar air, conditioned
by the distribution coefficient of acetone between plasma
and air at the body temperature.
The characteristic odor of the breath of patients showing symptoms of diaLetic coma and the fact that this odor is due atleast in part to acet’one. Although most of workers who have contributed
to this subject have devoted their attention to the excretion
of these substances by the kidneys but there have been many
observations on the excretion of acetone by the lungs.

i am partly right🙂
 
Can anybody explain
Acetylcholine is released at all of the following junctions expect
1-sympthatic ganglia
2-parasympthatic ganglia
3-somataic efferts to skeletal muscles
4-terminal sympathetic fibers to heart (ans)
5-terminal sympathetic fibers to adrenal medulla
Why can't the ans be 5?

adrenal medulla is just like ganglion(modified nerve tissue). preganglionic sympathetic fibers innervate the medulla directly and release Acetylcholine....hope this helps!
 
There was a question in previous papers that showed that hypothyroidism would cause postive protien balance. Positive protein balance is due to protein synthesis ( if i am right).
If thyroid has both protein synthesis and degradation function how can it alter protein balance?.
 
Positive protien balance (may)is also be because of lack of protein degradation; and not only systhesis of new protein
 
As far I know
Nitrogen balance - the difference between the amount of nitrogen taken in and the amount excreted or lost.

If you excrete more nitrogen than you consume your body will break down muscle tissue to get the nitrogen it needs. (Negative nitrogen state)- catabolic state. Muscle loss occurs.

If you consume more nitrogen than you excrete you will be in an anabolic - muscle building - state (positive nitrogen state).

Positive protien balance (may)is also be because of lack of protein degradation; and not only systhesis of new protein
 
Can any one answer them and explain me
Which cardivascular changes occur when changing from standing to supine position
1- decrease in contractibility
2- decrease in total peripheral resistance
3- dilation of large veins
4- increase in pulse (ans)
5- increase in renal blood flow

A 28 year old male with itravenous drug user present with symptons of infective endocarditis. IE is caused by
1-Enterococci
2-Strep pyogenus
3-Strep pneumonia
4-Staph aureus (ans)
5-Pseud aeruginosa

Velocity of nerve impulse in sensory nerve fibers is related to
1-intensity of stimulus
2-presence of myelin sheath
3-nature of applied stimulus
4-cross section area of axon (ans)
I think ans should be both 2 and 4?

10 year old boy with itch, vesicular rashes on face and trubk contains mixture of lesions of macules, papules and vesicles
1- Herpes I
2- HerpesII
3-Varicella
4-Shingeles
5- Measles
 
A 28 year old male with itravenous drug user present with symptons of infective endocarditis. IE is caused by
1-Enterococci
2-Strep pyogenus
3-Strep pneumonia
4-Staph aureus (ans)
5-Pseud aeruginosa
staphy aureus is usually present in the epidermis, iv cathers seen in Iv drug abusers.causing ABE. It is virulent hence cause damage to rt heart .emboli to lungs.if i am not wrong

Velocity of nerve impulse in sensory nerve fibers is related to
1-intensity of stimulus
2-presence of myelin sheath
3-nature of applied stimulus
4-cross section area of axon (ans)
I think ans should be both 2 and 4?
velocity increases in myelinated fibers with cross section. both are interconnected .can anybody explain??


10 year old boy with itch, vesicular rashes on face and trubk contains mixture of lesions of macules, papules and vesicles
1- Herpes I
2- HerpesII
3-Varicella
4-Shingeles
5- Measles[/QUOTE]
ithink it is varicella,dont now abt measles.shingles is recurrent.herpes occurs in children but only to oral region. herpes 2 in adults.
 
A 28 year old male with itravenous drug user present with symptons of infective endocarditis. IE is caused by
1-Enterococci
2-Strep pyogenus
3-Strep pneumonia
4-Staph aureus (ans)
5-Pseud aeruginosa
staphy aureus is usually present in the epidermis, iv cathers seen in Iv drug abusers.causing ABE. It is virulent hence cause damage to rt heart .emboli to lungs.if i am not wrong

Velocity of nerve impulse in sensory nerve fibers is related to
1-intensity of stimulus
2-presence of myelin sheath
3-nature of applied stimulus
4-cross section area of axon (ans)
I think ans should be both 2 and 4?
velocity increases in myelinated fibers with cross section. both are interconnected .can anybody explain??


10 year old boy with itch, vesicular rashes on face and trubk contains mixture of lesions of macules, papules and vesicles
1- Herpes I
2- HerpesII
3-Varicella
4-Shingeles
5- Measles
ithink it is varicella,dont now abt measles.shingles is recurrent.herpes occurs in children but only to oral region. herpes 2 in adults.[/QUOTE]

hi
ur answer is right..its varicella
the clinical picture of measles and varicella is almost same
in both lesions can involve face and body
but,varicella lesions are itchy and measles lesions are usually not
also, chances of macules,pappules and vesicle formation is more in varicella
 
ur answer is right..its varicella
the clinical picture of measles and varicella is almost same
in both lesions can involve face and body
but,varicella lesions are itchy and measles lesions are usually not
also, chances of macules,pappules and vesicle formation is more in varicella[/QUOTE]
thanks for good explanation
 
Thanks ! Both of you for your explanation.

ur answer is right..its varicella
the clinical picture of measles and varicella is almost same
in both lesions can involve face and body
but,varicella lesions are itchy and measles lesions are usually not
also, chances of macules,pappules and vesicle formation is more in varicella
thanks for good explanation[/QUOTE]
 
Can any one answer them and explain me
Which cardivascular changes occur when changing from standing to supine position
1- decrease in contractibility
2- decrease in total peripheral resistance
3- dilation of large veins
4- increase in pulse (ans)
5- increase in renal blood flow

reduced venous blood cause cardiac center activation which cause increased pulse pressure i am not sure though. anybody any explanations??
 
are you sure the ans is increased pulse pressure , i thought it was decreased peripheral resistance
 
are you sure the ans is increased pulse pressure , i thought it was decreased peripheral resistance

blood pressure on changing posture is maintained by baroreceptors,which are regulated by magnitude and the stretch of the arterial wall.now this depends on the pulse pressure and the mean arterial pressure
so, i think answer shud be the pulse pressure

please correct me if i am wrong
 
you are right i was thinking about the gravity factor which is wrong ,so the ans is increased pulse pressure
 
blood pressure on changing posture is maintained by baroreceptors,which are regulated by magnitude and the stretch of the arterial wall.now this depends on the pulse pressure and the mean arterial pressure
so, i think answer shud be the pulse pressure

please correct me if i am wrong

you are right that posture change in blood pressure is maintained by baroreceptors but I am still confused with that.

Is the answer choice really pulse pressure? (One more thing, pulse or pulse pressure.) If pulse i agree the answer is pulse because pulse = heart rate.

But if it's pulse pressure, pulse pressure = systolic pressure - diastolic pressure. From standing to supine position, venous return increase, stroke volume increase, cardiac out put increase, systolic pressure increase, increase pulse pressure. So the most possible answer choice will be decrease peripheral resistance due to decrease vasoconstriction.How's that?😛
 
Sorry Guys. I mistake in reading increase and decrease in the answer choices.
if increase pulse pressure agree.
but if increase pulse the answer should be decrease peripheral resistance I think.
 
Sorry Guys. I mistake in reading increase and decrease in the answer choices.
if increase pulse pressure agree.
but if increase pulse the answer should be decrease peripheral resistance I think.

i think u r right,its decrease in peripheral resistance
from standing to supine position,theres slight decreases in systolic BP ans slight vasodlatation with decreased peripheral vascular resistance
pulse increases(tachycardia) on standing
 
i am realy confused about the ans now , but what i strongly feel now is that due to lack of gravitational force as on standing position .there will be decreased pulse ,decresed pulse pressure and also will lead to decrease peripheral resistance when in horizontal position ....any more ans. or views....
 
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