Part One Pathology

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foxydentist

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Hey guys I thought of arranging the forum a bit by making a thread for each section for NBDE part one.... so if any body got any questions or need help in or got new useful infromation wana share in Pathology writes here......... 🙂 hope that would make it easy for everybody planning to take part one soon .. (like me 😳 )
 
Hello guys, yesterday, I was looking breifly at the "pathology" part in "kaplan" book,, and I was surprised at seeing so many topics that I don't think the dentist should know about, I was wondering if anyone is done with this part to give us some advice!!! thanks in advance 🙂
 
pls ans this,

1.type of necrosis in pyogenic infections?
a.coagulative
b.caseous
c.gummatous
d.liquefactive
e.enzymatic fat

a or d ?

2.bone lesion with radiopaque structures radiating from periphery suggests which?
a.osteoblastoma
b.osteosarcoma
c.multiple myeloma
d.giant cell tumor
e.osteitis deformans

b ?


thanx..
 
thats a good question. Firstly i thought about A as the answer but pyogenic infection have pus that is also seen is liquefactaive necrosis . So the answer is D
pls ans this,

4.type of necrosis in pyogenic infections?
a.coagulative
b.caseous
c.gummatous
d.liquefactive
e.enzymatic fat

a or d ?

thanx..
 
1.type of necrosis in pyogenic infections?
a.coagulative
b.caseous
c.gummatous
d.liquefactive
e.enzymatic fat
d

2.bone lesion with radiopaque structures radiating from periphery suggests which?
a.osteoblastoma
b.osteosarcoma
c.multiple myeloma
d.giant cell tumor
e.osteitis deformans
b
 
I need help,,,,,,,,,,,,, NBDE part one review book,,,, I feel that it contain lots of information,, I was flipping through the chapters,, and it got many details.,. i am wondering,,, is it too much or its just the right amount we gota study 4 part one..Thanks
 
Which of the following is thought to be of MOST significance in the etiology of microangiopathy in an uncontrolled diabetic?
A. Genetics
B. Hypertension
C. Blood glucose levels
D. Hyperlipidemia and hypercholesterolemia
E. Direct effect of insulin deficiency on endothelium

Answer with explanation please. Thanks
 
Which of the following represents complication of the peptic ulcer that accounts for majority of the deaths
1.Bleeding
2. Perforation
3. Obstruction
4. malignancy

I checked everywhere, 1-3 are the complications but not the cause of death........anybody any idea @ this?

Thanks in advance.....

Ct
 
Which of the following represents complication of the peptic ulcer that accounts for majority of the deaths
1.Bleeding
2. Perforation
3. Obstruction
4. malignancy

I checked everywhere, 1-3 are the complications but not the cause of death........anybody any idea @ this?

Thanks in advance.....

Ct

hi,
for NBDE the answer is perforation.....sure.........concluded from all other threads posted for same question

pls ans below question:

thymectomy results in what????????
 
hi,
for NBDE the answer is perforation.....sure.........concluded from all other threads posted for same question

pls ans below question:

thymectomy results in what????????

impaired T cell maturation so deficiency in cell mediated immunity
 
[Originally Posted by confused tooth View Post
Which of the following represents complication of the peptic ulcer that accounts for majority of the deaths
1.Bleeding
2. Perforation
3. Obstruction
4. malignancy

I checked everywhere, 1-3 are the complications but not the cause of death........anybody any idea @ this?

Thanks in advance.....

Ct]



The book I referred mentions bleeding '' Peptic ulceration is the commonest cause of serious and life threatening gastrointestinal bleeding.''
 
hi,
for NBDE the answer is perforation.....sure.........concluded from all other threads posted for same question

pls ans below question:

thymectomy results in what????????
Reni and abstract dentist.......thanks for your answers.......

I am just prsying that I do not get this question on my exam........and even if I do, I am gonna do eenie-minne-mine-mo :laugh:

Ct
 
ya i know it is anaphylactic but could you explain?
http://www.rch.org.au/bloodtrans/adverse.cfm?doc_id=5323

I found this on google searching for "adverse consequences of ABO mismatch"
I agree this link is not very helpful, but all of them have given a big example of "erythroblastalis fetalis", so it has to be delayed hypersensitivity (Type 4 ) ...........I guess....

This is all I could reason for this.......

Hope this helps..........

CT
 
http://www.rch.org.au/bloodtrans/adverse.cfm?doc_id=5323

I found this on google searching for "adverse consequences of ABO mismatch"
I agree this link is not very helpful, but all of them have given a big example of "erythroblastalis fetalis", so it has to be delayed hypersensitivity (Type 4 ) ...........I guess....

This is all I could reason for this.......

Hope this helps..........

CT
thanks for info confused tooth even i think it is type 4 anyway thanks for your effort.
 
Cancer with least potential for metastasis to bone?
tongue,prostate,breast,pancreas,thyroid
is it prostrate cancer????
 
The answer is "Thyroid Gland". Thyroid related cancers may rarely spread to the bones.
 
pls ans following question:
1 whatis fluoride form in stomach?
2.calcium binding protein in saliva/?
3.during yawning which ligament affected?
4.during clenching which muscle is affected?
5.clenching is affected by which muscle? choice is b/w temporalis and medial pterygoid............
 
Cancer with least potential for metastasis to bone?
tongue,prostate,breast,pancreas,thyroid
is it prostrate cancer?

ans is tongue
The most common cancers to spread to the bones are prostate, lung and breast cancer.
 
i am not sure about the answer

calcium binding protein in saliva/
albumin

during yawning which ligament affected
tm lig. (as yawning can cause dislocation)

during clenching which muscle is affected
temporalis
 
PNEUNOCYSTIS CARINII WHICH CAUSES PNEUMOCYSTOSIS ??

👍 IS A FUNGAL DISEASE OR A PARASITIC DISEASE
 
Q which represents an inflammatory exudate with few cells and is composed of watery low protein fluid?
serous
fibrous
purulent
fibrinous

is it serous exudate?

Q the genetic hypothesis of cancer implies that genetic damage to single progenitor cells ultimately results in tumor mass .which of the following describes cells that compose tumor mass?
monoclonal
heterogenous
void of antigens
contact inhibited
???
 
pls ans following question:
1 whatis fluoride form in stomach?
2.calcium binding protein in saliva/?
3.during yawning which ligament affected?
4.during clenching which muscle is affected?
5.clenching is affected by which muscle? choice is b/w temporalis and medial pterygoid............

i think
1> floride is in form hydrofloric acid in stomach and is absorbed in intestine ..intestine has max adverse effects to floride
also since fl is more electronegative than ca and replace it leading to hypocalcemia
2> calcium bindin protein in saliva
is albumin

3>during yawning tmj is affected

4>during clenching masseter muscle damaged
 
i think
1> floride is in form hydrofloric acid in stomach and is absorbed in intestine ..intestine has max adverse effects to floride
also since fl is more electronegative than ca and replace it leading to hypocalcemia
2> calcium bindin protein in saliva
is albumin

3>during yawning tmj is affected

4>during clenching masseter muscle damaged

thnks indian bds,
can u pls tell how massetor is afected while clenching?? one of our fellow members answered temporalis n now i m confused......

and for all of u....... the ans to second question
calcium binding protein in saliva.....it is proline rich proteins........
 
thnks indian bds,
can u pls tell how massetor is afected while clenching?? one of our fellow members answered temporalis n now i m confused......

and for all of u....... the ans to second question
calcium binding protein in saliva.....it is proline rich proteins........

It seems to me that clenching affects temporalis based on "wheelers". This topic is discussed in the book but I read it a little bit while ago...
 
Q which represents an inflammatory exudate with few cells and is composed of watery low protein fluid?
serous
fibrous
purulent
fibrinous

is it serous exudate?

Q the genetic hypothesis of cancer implies that genetic damage to single progenitor cells ultimately results in tumor mass .which of the following describes cells that compose tumor mass?
monoclonal
heterogenous
void of antigens
contact inhibited
???


Q1. yeah the ans. is serous exudate
Q2. monoclonal( all the cancer cells come from a single progenitor so they are monoclonal in the sense that they have the same genetic information)
 
Cancer with least potential for metastasis to bone?
tongue,prostate,breast,pancreas,thyroid
is it prostrate cancer?

ans is tongue
The most common cancers to spread to the bones are prostate, lung and breast cancer.

yeap agree with Dr.neha
 
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