With plavix you'll have problems forming the platelet plug. But with some local measures (pressure and anything else you want to throw into the socket) you'll get past the initial gush and the fibrin clot will form pretty quickly.
In contrast to Plavix (or aspirin aggrenox, etc), the (theoretical) problem with blood thinners that work on the fibrin cascade (heparin, coumadin, etc) is that you form your initial plug but the clot is slow to mature because the drugs are inhibiting fibrin formation. So in these patients, you might seem like you have good hemostasis and then the patient rubs the area with their tongue and GUSH they start bleeding again. And this might happen after they've gone home... oops!
In practice, I've never stopped plavix or aspirin. We took a tooth out on someone with platelets at 35. However, I've refused to do surgery with high INRs (north of 3 for elective perio surgery and north of 4 for single, painful tooth extraction) and I have bridged with heparin. But understanding WHY you're doing something is important - this way your decision can be case-specific and not dogmatic.