Because we learned it in class, so I know its true.
Plus it's not hard to reason out, AV node is the site with the highest delay, if there is a defect there you an expect the PR interval to lengthen.
Since Mobitz type-II involves random drops of QRS complexes not proceeded by changes in the PR interval you can assume there is no problem making it through the AV node, only a problem with the final conduction to the ventricles. Hence, His-Purkinje defect.
Im pretty sure it was explained better either in FA, or Rx though.