This is characteristically produced by noninfectious inflammatory diseases, such as rheumatic fever, SLE, and scleroderma, tumors, and uremia
Fibrinous and Serofibrinous Pericarditis
Common causes include acute MI (recall Fig. 12-19D), the postinfarction (Dressler) syndrome (probably an autoimmune condition appearing several weeks after an MI), uremia, chest radiation, rheumatic fever, SLE, and trauma. A fibrinous reaction also follows routine cardiac surgery.
That seems like a difficult clinical distinction.
From what I remember during my studies, you're less likely to encounter a question that describes a patient with SLE, pericarditis, and an effusion that asks for the type of pericarditis than you are to see a question describing a young patient with chest pain relieved by leaning forward, a friction rub on auscultation that persists through the patient holding their breath, and then asks for the diagnosis.]I'd be interested to hear from someone who knows how to, if possible, make a clinical distinction between the two.
How about serous vs. fibrinous? Both are a form of pericarditis, so everything you mentioned at first will be present in both. Fibrinous can be associated with an effusion, and, well, serous obviously can, too. So, everything you mentioned in the second portion may also be present.
If I understood Phloston's question, how can we differentiate them from one another?
I've actually had a Kaplan QBook question that asked about the type of pericarditis following an MI. Both immediately after and weeks later (Dressler's) are fibrinous.