So what do you really think of Travell's trigger point work

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Josh L.Ac.

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Background: Travell's two-book set is the granddaddy of all collections for those interested in biomedical acupuncture. Most of the medical acupuncturists I've spoken with use it more than any other style of acupuncture.




I was looking through volume 2 again today and I started to wonder what physicians trained in interventional pain management honestly thought about it.


So for those of you that are familiar with her work (whether or not you perform acupuncture or point injections), what do you honestly think of it? If I go the FNP route, acupuncture + point injections [plus medications] will be about the extent of what I can do within my scope, so I was wondering how effective it was in the context of interventional pain management.

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Background: Travell's two-book set is the granddaddy of all collections for those interested in biomedical acupuncture. Most of the medical acupuncturists I've spoken with use it more than any other style of acupuncture.

I was looking through volume 2 again today and I started to wonder what physicians trained in interventional pain management honestly thought about it.

So for those of you that are familiar with her work (whether or not you perform acupuncture or point injections), what do you honestly think of it? If I go the FNP route, acupuncture + point injections [plus medications] will be about the extent of what I can do within my scope, so I was wondering how effective it was in the context of interventional pain management.

Complex question. Trigger point injections (regardless of where you do them and with what magic elixir) are simply percutaneous physical modality interventions. The analgesic properties of all physical modalities are grounded in the physiology of counter-irritation. Let's form a circle, hold hands, and chant it in unison, "The-analgesic-properties-of-all-physical-modalities-are-grounded-in-the-physiology-of-counter-irritation...."

So, I'm not surprised that *SOME* people benefit from trigger point injections. For chronic pain, I think it is a palliative intervention at best. It is usually passive--i.e. Mrs. Jones comes in every 3 weeks and is needled with 1-3 gallons of local anesthetic mixed with some infintesimal amount of steroid (or even better Sarapin!) and then goes home. What have we accomplished? It doesn't seem like a good long-term solution for a long term problem.

Having said that, I incurred a cervical strain from poor weight lifting technique at the gym and couldn't "shake it off." I had some trigger point injections done, along with a little "osteopathic magic" and lo and behold...I felt a little better. So, maybe for acute problems, it may be of benefit.
 
Crazy voodoo BS.

But if it makes them feel better and they understand it lasts 1 hr to 2 weeks if they don't fix the underlying problem.

If the doc is using anything other than straight LA to ease the pain of the procedure- they are incurring more risk than the procedure is worth.
 
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Crazy voodoo BS.

But if it makes them feel better and they understand it lasts 1 hr to 2 weeks if they don't fix the underlying problem.

If the doc is using anything other than straight LA to ease the pain of the procedure- they are incurring more risk than the procedure is worth.

Don't tell them it will only last 1-2 hours! These are largely placebos, with a little bit of end-organ treatment (as opposed to addressing the root cause).

If you tell drusso his injection was only supposed to last two hours, his pain will come back!
 
i like doing trigger point injections (strike that...i actually hate DOING them) to ease the pain enough for them to participate in physical therapy and actually fix the dysfunctional muscle firing (or at least that's what i think is the actual cause of them).

i do agree somewhat with steve and ampaphb though that most of the relief is placebo effect (like most of what pain medicine is). studies have shown that dry needling is just as effective (though will cause local muscle inflammation and have the patient in more pain that night). ischemic compression is just as effective but takes way more time. another study is read recently (i forget which one) showed the benefits of suggestive positive attitude for a procedure (meaning the doctor being confident a procedure will work and expressing that to the patient made a big difference in the actual effects). i think that placebo suggestion of benefits should be multilayered in an office setting, with the other staff (PTs, nurses, etc) saying great things about the MD and how well he helps his/her patients and giving a brief example of a recent patient (avoiding HIPAA violations of course) that feels like a new man after "that" procedure.
 
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i like doing trigger point injections (strike that...i actually hate DOING them) to ease the pain enough for them to participate in physical therapy and actually fix the dysfunctional muscle firing (or at least that's what i think is the actual cause of them).

i do agree somewhat with steve and ampaphb though that most of the relief is placebo effect (like most of what pain medicine is). studies have shown that dry needling is just as effective (though will cause local muscle inflammation and have the patient in more pain that night). ischemic compression is just as effective but takes way more time. another study is read recently (i forget which one) showed the benefits of suggestive positive attitude for a procedure (meaning the doctor being confident a procedure will work and expressing that to the patient made a big difference in the actual effects). i think that placebo suggestion of benefits should be multilayered in an office setting, with the other staff (PTs, nurses, etc) saying great things about the MD and how well he helps his/her patients and giving a brief example of a recent patient (avoiding HIPAA violations of course) that feels like a new man after "that" procedure.

I've had procedures that just were too technically difficult that I would be hurting for a few days and cringing about seeing them back in follow-up. I tell them things were tough but we got the medicine on the targets and it all looked good. THey usually come back without as much as an ache. I love the elderly- they can get better no matter what you do- probably moreso because they want to.
 
I love the elderly- they can get better no matter what you do- probably moreso because they want to.

yep. i had a 71yo guy at the VA today that got 5.5 months of great relief from 5 trigger point injections back in february. he had neck and referred arm pain after multiple cervical laminectomies and fusions and this just seemed to do the trick. and yes, we used 40mg depomedrol (and 1cc at each TP of 0.5% marcaine). and i don't care if that was 100% placebo or not. that veteran doesn't care either. he's a believer so i'm willing to delivery the goods. 🙂
 
She was past eighty when the first volume of her grand opus, Myofascial Pain & Dysfunction: The Trigger Point Manual was published, and past ninety when the second volume appeared. The octagenarian/nonagenarian approach to pain may appeal to many, but if there were significant science behind this approach, more of us would be interested.... Much of myofascial pain/trigger poin therapy is more related to meridian theory rather than modern disease state quantification/treatment. Travell has been dead for 11 years. My interest in this arena died before she did.
 
She was past eighty when the first volume of her grand opus, Myofascial Pain & Dysfunction: The Trigger Point Manual was published, and past ninety when the second volume appeared. The octagenarian/nonagenarian approach to pain may appeal to many, but if there were significant science behind this approach, more of us would be interested.... Much of myofascial pain/trigger poin therapy is more related to meridian theory rather than modern disease state quantification/treatment. Travell has been dead for 11 years. My interest in this arena died before she did.

wait. are you saying trigger points don't exist?? i've treated a TON of headaches that are exacerbated during the exam by pressing on a trigger point and the patient stating that reproduces the headache location exactly. whether you inject, or dry needle, or ischemic compression, or adjust the z-joints, whatever it takes to get rid of those trigger points works for their pain relief. my observation of good benefit from treating trigger points stops at headache and shoulder pain relief. and some docs inject the trigger points with botox...i think that's just WAY overkill and too expensive.
 
Arch Phys Med Rehabil. 2008 Jun;89(6):1169-76.

A systematic, critical review of manual palpation for identifying myofascial
trigger points: evidence and clinical significance.

Myburgh C, Larsen AH, Hartvigsen J.

Institute of Sports Science and Clinical Biomechanics, University of Southern
Denmark. [email protected]

OBJECTIVE: To determine the reproducibility of manual palpation in identifying
trigger points based on a systematic review of available literature. DATA
SOURCES: Medline (1965-2007), CINHAL (1982-2007), ISI Web of Science (1945-2007),
and MANTIS (1966-2007) databases and reference lists of articles. STUDY
SELECTION: Reproducibility studies relating to identification and diagnosis of
trigger points through palpation. Acceptable studies were required to
specifically consider either inter- or intrarater reliability of trigger point
identification through manual palpation and include kappa statistics as part of
their statistical assessment. DATA EXTRACTION: Three independent reviewers
considered the studies for inclusion and rated their methodologic quality based
on the Standards for Reporting of Diagnostic Accuracy guidelines for the
reporting of diagnostic studies. DATA SYNTHESIS: Eleven studies were initially
included; however, 5 were subsequently excluded based on the inclusion and
exclusion criteria. Only 2 studies were judged to be of high quality, and the
level of evidence criteria suggested that, at best, moderate evidence could be
found from which to make pronouncements on the literature. Only local tenderness
of the trapezius (kappa range, .15-.62) and pain referral of the gluteus medius
(kappa range, .298-.487) and quadratus lumborum (kappa range, .36-.501) were
found to be reproducible. CONCLUSIONS: The methodologic quality of the majority
of studies for the purpose of establishing trigger point reproducibility is
generally poor. More high-quality studies are needed to comment on this
procedure. Clinicians and scientists are urged to move toward simpler, global
assessments of patient status.

Eur J Pain. 2008 Apr 3. [Epub ahead of print]

Acupuncture and dry needling in the management of myofascial trigger point pain:
A systematic review and meta-analysis of randomised controlled trials.

Tough EA, White AR, Cummings TM, Richards SH, Campbell JL.

Primary Care, Peninsula Medical School, Universities of Exeter and Plymouth, Room
N32, ITTC Building, Tamar Science Park, Plymouth PL6 8BX, UK.

Pain from myofascial trigger points is often treated by needling, with or without
injection, although evidence is inconclusive on whether this is effective. We
aimed to review the current evidence on needling without injection, by conducting
a systematic literature review. We searched electronic databases to identify
relevant randomised controlled trials, and included studies where at least one
group were treated by needling directly into the myofascial trigger points, and
where the control was either no treatment, or usual care; indirect local dry
needling or some form of placebo intervention. We extracted data on pain, using
VAS scores as the standard. Seven studies were included. One study concluded that
direct dry needling was superior to no intervention. Two studies, comparing
direct dry needling to needling elsewhere in the muscle, produced contradictory
results. Four studies used a placebo control and were included in a
meta-analysis. Combining these studies (n=134), needling was not found to be
significantly superior to placebo (standardised mean difference, 14.9 [95%CI,
-5.81 to 33.99]), however marked statistical heterogeneity was present
(I(2)=88%). In conclusion, there is limited evidence deriving from one study that
deep needling directly into myofascial trigger points has an overall treatment
effect when compared with standardised care. Whilst the result of the
meta-analysis of needling compared with placebo controls does not attain
statistically significant, the overall direction could be compatible with a
treatment effect of dry needling on myofascial trigger point pain. However, the
limited sample size and poor quality of these studies highlights and supports the
need for large scale, good quality placebo controlled trials in this area.

Clin J Pain. 2007 Mar-Apr;23(3):278-86.

Variability of criteria used to diagnose myofascial trigger point pain
syndrome--evidence from a review of the literature.

Tough EA, White AR, Richards S, Campbell J.

Peninsula Medical School, Universities of Exeter and Plymouth, UK.
[email protected]

OBJECTIVES: The aim of the literature review was to investigate the criteria
adopted by "experts" to diagnose myofascial trigger point (MTrP) pain syndrome.
Experts were defined as being either researchers investigating MTrP pain syndrome
or the "authority" the researchers cited as a source of reference for MTrP pain
syndrome diagnosis. METHODS: We searched electronic databases to identify
relevant empirical research (excluding studies not in English and those relating
to dental pathology). Of 607 possibly relevant publications 93 met our inclusion
criteria. We recorded (1) the individual criterion and criteria combinations used
to diagnose MTrP pain syndrome; (2) the cited "authoritative" publications and
(3) the criteria recommended by the authoritative publications as being essential
for MTrP pain syndrome diagnosis. RESULTS: The review identified 19 different
diagnostic criteria. The 4 most commonly applied criteria were: "tender spot in a
taut band" of skeletal muscle, "patient pain recognition," "predicted pain
referral pattern," and "local twitch response." There was no consistent pattern
to the choice of specific diagnostic criteria or their combinations. However, one
pair of criteria "tender point in a taut band" and "predicted or recognized pain
referral" were used by over half the studies. The great majority of studies cited
publications by Travell and more recently Simons as a principal authoritative
source for MTrP pain syndrome diagnosis, yet most of these studies failed to
apply the diagnostic criteria as described by these authorities. DISCUSSION: We
conclude that there is as yet limited consensus on case definition in respect of
MTrP pain syndrome. Further research is needed to test the reliability and
validity of diagnostic criteria. Until reliable diagnostic criteria have been
established, there is a need for greater transparency in research papers on how a
case of MTrP pain syndrome is defined, and claims for effective interventions in
treating the condition should be viewed with caution.
 
those studies are more of evidence of LACK of evidence regarding trigger point injections...rather than evidence of needling/injections not being effective. BUT, thanks for bringing this to my attention. i really didn't know there was such a lack of evidence regarding TP injections. and for the record, if i ran the world (vote for faubel 2032) a trigger point would be a tender point in a muscle's taut band that causes point tenderness and REFERRAL of pain. and it would only be treated if symptomatically causing pain in the distribution of the patient's primary complaint.

i should get more literature and do a talk for the other residents (ass. chief duties).
 
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there are plenty of reports that show the histologic changes at trigger points are non-specific. i think the best way to get specific histologic changes at a TP is to put a needle there, which many love to do for some reason.

the whole theory is a pretty much a waste of time and energy as others noted. if you actually try to read simon and travell, you will have a hard time getting past the first few chapters if you have a critical mind.

you may find in your future investigations that the TPs represent referred pains.
 
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The lack of studies showing effectiveness in systematic reviews is indicative these procedures do not work and are not reproducible. There are a dearth of studies on histological changes in TP because most have shown there are no histological changes. Microscopic analysis of TPs show no differences between surrounding tissue. There is one study demonstrating an increase in substance P in TP and several others showing single fiber changes on EMG, and one showing MRI changes in the cross-sectional area of an affected muscle, but these all suggest hypertrophy of the muscle rather than any specific disease.
 
i would say that the average TP works about 3 weeks.... is this placebo?

i don't have an answer.
 
I recall an older German rabbit study showing decreased O2 tension in the muscle at the tender area. Don't know what that means clinically or if that occurs at random samplings.

Could put it off as risk vs benefit- only a few case reports of things gone awry- IT injection by a FP doing trap TP's comes to mind. It just seems like a waste of time unless the patient specifically requests it. Then the placebo effect > therapeutic effect for the procedure.
 
1: Arch Phys Med Rehabil. 2008 Jan;89(1):16-23. Links

Comment in: Arch Phys Med Rehabil. 2008 Jan;89(1):157-9. Biochemicals associated with pain and inflammation are elevated in sites near to and remote from active myofascial trigger points.

Shah JP, Danoff JV, Desai MJ, Parikh S, Nakamura LY, Phillips TM, Gerber LH.
Rehabilitation Medicine Department, National Institutes of Health, Clinical Center, Bethesda, MD 20892, USA. [email protected]
OBJECTIVES: To investigate the biochemical milieu of the upper trapezius muscle in subjects with active, latent, or absent myofascial trigger points (MTPs) and to contrast this with that of the noninvolved gastrocnemius muscle. DESIGN: We used a microanalytic technique, including needle insertions at standardized locations in subjects identified as active (having neck pain and MTP), latent (no neck pain but with MTP), or normal (no neck pain, no MTP). We followed a predetermined sampling schedule; first in the trapezius muscle and then in normal gastrocnemius muscle, to measure pH, bradykinin, substance P, calcitonin gene-related peptide, tumor necrosis factor alpha, interleukin 1beta (IL-1beta), IL-6, IL-8, serotonin, and norepinephrine, using immunocapillary electrophoresis and capillary electrochromatography. Pressure algometry was obtained. We compared analyte concentrations among groups with 2-way repeated-measures analysis of variance. SETTING: A biomedical research facility. PARTICIPANTS: Nine healthy volunteer subjects. INTERVENTIONS: Not applicable. MAIN OUTCOME MEASURES: Preselected analyte concentrations. RESULTS: Within the trapezius muscle, concentrations for all analytes were higher in active subjects than in latent or normal subjects (P<.002); pH was lower (P<.03). At needle insertion, analyte concentrations in the trapezius for the active group were always higher (pH not different) than concentrations in the gastrocnemius muscle. At all times within the gastrocnemius, the active group had higher concentrations of all analytes than did subjects in the latent and normal groups (P<.05); pH was lower (P<.01). CONCLUSIONS: We have shown the feasibility of continuous, in vivo recovery of small molecules from soft tissue without harmful effects. Subjects with active MTPs in the trapezius muscle have a biochemical milieu of selected inflammatory mediators, neuropeptides, cytokines, and catecholamines different from subjects with latent or absent MTPs in their trapezius. These concentrations also differ quantitatively from a remote, uninvolved site in the gastrocnemius muscle. The milieu of the gastrocnemius in subjects with active MTPs in the trapezius differs from subjects without active MTPs.
 
The same study also showed trapezius levels for each of the cytokines were higher than the gastrocs levels in active, latent, and normal groups, therefore the control used (gastrocs) was not all that useful. It also showed the gastrocs levels were higher for active>latent>normal groups which suggests this is a systemic response to pain rather than a focal response.
Finally, it would have been far more useful to compare TP in the trapezius with adjacent non-trigger points in the trapezius. That is the only way to demonstrate focal pathology exists within a trigger point. They did not do this type of study, therefore it appears there results support a systemic rather than focal response to pain....
 
The same study also showed trapezius levels for each of the cytokines were higher than the gastrocs levels in active, latent, and normal groups, therefore the control used (gastrocs) was not all that useful. It also showed the gastrocs levels were higher for active>latent>normal groups which suggests this is a systemic response to pain rather than a focal response.
Finally, it would have been far more useful to compare TP in the trapezius with adjacent non-trigger points in the trapezius. That is the only way to demonstrate focal pathology exists within a trigger point. They did not do this type of study, therefore it appears there results support a systemic rather than focal response to pain....


i agree with the above, but your statement:

"There are a dearth of studies on histological changes in TP because most have shown there are no histological changes"

i cant fully support. something's going on in those trigger points. we may not know exactly what, but somethings going on. since you cant excise, mash up, and analyze a human's trapezius muscle without the patient getting a bit upset, the methods used in this study give us a decent look at the physiology of a trigger point. jay shah usually does some wacky myofascial talk at the AAPMR. i assume he'll do one this year and talk about the above study.
 
I agree something is present, but histologically and for the most part biochemically, there seems to be no specific abnormality that is not present in other chronic pain conditions. In uncontrolled single fiber EMG testing there is some evidence that the muscle is abnormal, but without controls, how does one know.... The discriminatory tactile ability of our digital exam to pick up these "taut bands" is very questionable given that we may well be perceiving fascia, subcutaneous tissue, etc as these "taut bands". In the obese, forget it...
I do inject them when severe....but frankly I don't know the pathology of what I am injecting
 
I agree something is present, but histologically and for the most part biochemically, there seems to be no specific abnormality that is not present in other chronic pain conditions.

That hasn't stopped the fibromyalgia lobby. We even have FDA-approved drugs for it.

I used to do a lot of TPIs 10-12 years ago. I hardly do any now unless there is a really severe acute problem and I want to put out the fire. This was not a deliberate decision, I just sort of stopped doing them. I still have half a bottle of Sarapin of indeterminate age. I am saving it for my great-grandchildren to sell on eBay.
 
That hasn't stopped the fibromyalgia lobby. We even have FDA-approved drugs for it.

I used to do a lot of TPIs 10-12 years ago. I hardly do any now unless there is a really severe acute problem and I want to put out the fire. This was not a deliberate decision, I just sort of stopped doing them. I still have half a bottle of Sarapin of indeterminate age. I am saving it for my great-grandchildren to sell on eBay.

so what are you doing with those patients that you previously would've done TP injections on??
 
well --- then you guys can help me with this dilemma.

40 yo lady w/ HORRIBLE myofascial pain of the neck after bony tumor resection from cervical spine followed by Anterior/Posterior fusion... Her cervical para-spinal and traps are LIKE CEMENT.

NOTHING pharmacologic works other than nortriptyline/zanaflex at night-time - too sedating during the day

PT helps the pain from getting worse - if she doesn't get PT at least once a week she is a crying mess.

She has had every procedure known to man at multiple pain clinics.

When she gets TPs every 4 weeks, she can function, can take care of her family, can work, is not depressed, etc.... and her muscles feel softer

When her insurance drags its feet w/ auths (oh yeah baby - auths for TPIs), the opposite occurs... she becomes dysfunctional, lays in bed crying from pain, and her muscles feel like concrete...

any suggestions? i use 10 ml of lido 1% w/ 27g needles every 4 weeks...
 
If it works, I would continue doing it forever if need be, with the understanding the mechanism may be unknown. Lidocaine is the least expensive drug we can buy and for the cost of a 10 cent syringe and a 15 cent needle plus 2 minutes of your time, you can make her life more tolerable, even if it is for free. Other options: try 6-10% lidocaine injections (very small volume) in the same location.
 
well --- then you guys can help me with this dilemma.

40 yo lady w/ HORRIBLE myofascial pain of the neck after bony tumor resection from cervical spine followed by Anterior/Posterior fusion... Her cervical para-spinal and traps are LIKE CEMENT.

NOTHING pharmacologic works other than nortriptyline/zanaflex at night-time - too sedating during the day

PT helps the pain from getting worse - if she doesn't get PT at least once a week she is a crying mess.

She has had every procedure known to man at multiple pain clinics.

When she gets TPs every 4 weeks, she can function, can take care of her family, can work, is not depressed, etc.... and her muscles feel softer

When her insurance drags its feet w/ auths (oh yeah baby - auths for TPIs), the opposite occurs... she becomes dysfunctional, lays in bed crying from pain, and her muscles feel like concrete...

any suggestions? i use 10 ml of lido 1% w/ 27g needles every 4 weeks...

DO NOT TRY THIS:

Try this- inject her with NSS instead of lidcaine and gauge the response.


She may have unrealistic expectations and could benefit from clinical psychology for CBT, coping strategies. TENS, Biofeedback. It would be really neat to try dry needling vs sham needling (unsure how that works)
How about hybresis patches from PT?

Is there any healthcare personnel at home. a 30G 1/2" needle can be easily taught to an LPN for injections at home. I have a few patients who have perfromed SPG's at home and a few more who have their spouses do cervical paraspinal Marcaine injections for migraine.
 
Excellent discussion. I agree in large part with the lack of evidence, and I believe that the majority of TPI result in a placebo response. That said, I still do a couple of them a week to put out fires. I'm not going to get up too high on a soapbox in their support, but I'm quite certain that TPI's have saved several ED trips, and I'm reasonably certain that in using them, I've prevented patients from being initiated on or escalations of chronic opioid therapy.

And they are "reasonably safe", as long as you realize that they're not "completely safe" and aren't an idiot. I did see a patient who got 24 TPIs for thoracic pain and a PTX, picked up after she left this docs clinics who told her the SOB was nerves and would get better after she left.
 
I did see a patient who got 24 TPIs for thoracic pain and a PTX, picked up after she left this docs clinics who told her the SOB was nerves and would get better after she left.

steve, that doctor must have been a chiropractor right? overt incompetence surely doesn't occur with MDs. ;-) just kidding steve. :laugh:
 
well --- then you guys can help me with this dilemma.

40 yo lady w/ HORRIBLE myofascial pain of the neck after bony tumor resection from cervical spine followed by Anterior/Posterior fusion... Her cervical para-spinal and traps are LIKE CEMENT.

NOTHING pharmacologic works other than nortriptyline/zanaflex at night-time - too sedating during the day

PT helps the pain from getting worse - if she doesn't get PT at least once a week she is a crying mess.

She has had every procedure known to man at multiple pain clinics.

When she gets TPs every 4 weeks, she can function, can take care of her family, can work, is not depressed, etc.... and her muscles feel softer

When her insurance drags its feet w/ auths (oh yeah baby - auths for TPIs), the opposite occurs... she becomes dysfunctional, lays in bed crying from pain, and her muscles feel like concrete...

any suggestions? i use 10 ml of lido 1% w/ 27g needles every 4 weeks...

botox...
 
steve, that doctor must have been a chiropractor right? overt incompetence surely doesn't occur with MDs. ;-) just kidding steve. :laugh:

Depends on whether it was before or after the coding changes.

Likely an FP. Probably the same one that got IT on a trap TrP.

Ann Emerg Med. 1998 Oct;32(4):506-8.
Intrathecal injection: unusual complication of trigger-point injection therapy.Nelson LS, Hoffman RS.
New York University Medical Center/Bellevue Hospital Center, NY, USA. [email protected]

Trigger-point injection therapy is a common procedure in primary care medicine and emergency medicine and is generally considered safe. A 28-year-old woman experienced respiratory depression and hemiplegia after the injection of a superficial trapezius trigger point. The patient required emergency tracheal intubation for ventilatory support. Computed tomography of her head revealed pneumocephalus. She recovered fully over the course of 24 hours. Intrathecal injection during a trigger-point injection is a previously unreported complication of trigger-point injection therapy.
 
There is an anesthesiologist in my area without any pain training other than going to a few annual meetings, who opened a "pain clinic" a few years back. This guy gives TP injections with steroids virtually on every visit since it nets him more money, and then does deep spinal injections on these same patients several times a year. I recently acquired one of his patients that had had 40 injections over a year and a half....
TPI can be grossly overused in those that value $$$ more than patient welfare.
 
she has had botox, she has had acupuncture/pressure/reiki, she has had CBT/biofeedback, she has tried all the usual anti-depressants/anxiolytics, she sees a counselor, she has tried the TENS

interesting concept of letting a patient self-inject... it would have to be a very short needle, as some patients think deeper is better and the next thing you know they drop a lung or hit a part of the cervical spine... 🙁
 
well --- then you guys can help me with this dilemma.

40 yo lady w/ HORRIBLE myofascial pain of the neck after bony tumor resection from cervical spine followed by Anterior/Posterior fusion... Her cervical para-spinal and traps are LIKE CEMENT.

NOTHING pharmacologic works other than nortriptyline/zanaflex at night-time - too sedating during the day

PT helps the pain from getting worse - if she doesn't get PT at least once a week she is a crying mess.

She has had every procedure known to man at multiple pain clinics.

When she gets TPs every 4 weeks, she can function, can take care of her family, can work, is not depressed, etc.... and her muscles feel softer

When her insurance drags its feet w/ auths (oh yeah baby - auths for TPIs), the opposite occurs... she becomes dysfunctional, lays in bed crying from pain, and her muscles feel like concrete...

any suggestions? i use 10 ml of lido 1% w/ 27g needles every 4 weeks...

does her neck hurt?....when we sprain a finger we make a splint with two popsicle sticks. when the spine hurts i have found that the muscles will contract to limit motion like a natural splint, and the pain is usually from the facets. did she have a C4-5 fusion?
 
i have done cervical facet RF - she has had good relief of her upper neck pain and resolution of her headaches...... her fusion is from C4 to T1.
 
There is an anesthesiologist in my area without any pain training other than going to a few annual meetings, who opened a "pain clinic" a few years back. This guy gives TP injections with steroids virtually on every visit since it nets him more money, and then does deep spinal injections on these same patients several times a year. I recently acquired one of his patients that had had 40 injections over a year and a half....
TPI can be grossly overused in those that value $$$ more than patient welfare.

There's a guy like that in my area. He bills the shots as facet injections and MBBs. Never saw an EOB from this guy for an office visit that was less than $600. I got tired of seeing the AVN, cushingoid faces, etc from his practice. The last straw was when he was doing weekly steroid injections on a pt with Ehler-Danlos.

A few phone calls were made and now he is being proctoscoped by several state and federal agencies. I also notify the insurance companies and include copies of relevant records along with annotations.
 
There is an anesthesiologist in my area without any pain training other than going to a few annual meetings, who opened a "pain clinic" a few years back. This guy gives TP injections with steroids virtually on every visit since it nets him more money, and then does deep spinal injections on these same patients several times a year. I recently acquired one of his patients that had had 40 injections over a year and a half....
TPI can be grossly overused in those that value $$$ more than patient welfare.


I recently reviewed the U.S. medicare schedule of benefits and found it pretty surprising. $54 for office TP injections? Sounds pretty ridiculous to me.

Canadian MDs are paid $8 for a TP injection. I use this treatment to benefit the patient , not my wallet.
 
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Identification and Quantification of Myofascial Taut Bands
With Magnetic Resonance Elastography

Archives of Physical Medicine and Rehabilitation, Volume 88, Issue 12, December 2007, Pages 1658-1661
Qingshan Chen, MS, Sabine Bensamoun, PhD, Jeffrey R. Basford, MD, PhD, Jeffrey M. Thompson, MD,
Kai-Nan An, PhD
ABSTRACT. Chen Q, Bensamoun S, Basford JR, Thompson
JM, An K-N. Identification and quantification of myofascial
taut bands with magnetic resonance elastography. Arch
Phys Med Rehabil 2007;88:1658-61.
Objective: To explore the feasibility of using a new magnetic
resonance imaging (MRI) technique—magnetic resonance
elastography (MRE)—to identify and quantitate the nature
of myofascial taut bands.
Design: This investigation consisted of 3 steps. The first
involved proof of concept on gel phantoms, the second involved
numeric modeling, and the third involved a pilot trial on
2 subjects. Imaging was performed with a 1.5T MRI machine.
Shear waves were produced with a custom-developed acoustically
driven pneumatic transducer with gradient-echo image
collection gated to the transducer’s motion. Shear wave propagation
were imaged by MRE.
Setting: An MRI research laboratory.
Participants: Two women, one with a 3-year history of
myofascial pain and the other serving as the control.
Interventions: Not applicable.
Main Outcome Measures: MRE images, finite element analysis
calculations, and tissue and phantom stiffness determinations.
Results: Results of the phantom measurements, finite element
calculations, and study patients were all consistent with
the concept that taut bands are detectable and quantifiable with
MRE imaging. The findings in the subjects suggest that the
stiffness of the taut bands (9.00.9KPa) in patients with myofascial
pain may be 50% greater than that of the surrounding
muscle tissue.
Conclusions: Our findings suggest that MRE can quantitate
asymmetries in muscle tone that could previously only be
identified subjectively by examination.
Key Words: Magnetic resonance imaging; Myofascial pain;
Rehabilitation.
 
Identification and Quantification of Myofascial Taut Bands
With Magnetic Resonance Elastography

Archives of Physical Medicine and Rehabilitation, Volume 88, Issue 12, December 2007, Pages 1658-1661
Qingshan Chen, MS, Sabine Bensamoun, PhD, Jeffrey R. Basford, MD, PhD, Jeffrey M. Thompson, MD,
Kai-Nan An, PhD
ABSTRACT. Chen Q, Bensamoun S, Basford JR, Thompson
JM, An K-N. Identification and quantification of myofascial
taut bands with magnetic resonance elastography. Arch
Phys Med Rehabil 2007;88:1658-61.
Objective: To explore the feasibility of using a new magnetic
resonance imaging (MRI) technique—magnetic resonance
elastography (MRE)—to identify and quantitate the nature
of myofascial taut bands.
Design: This investigation consisted of 3 steps. The first
involved proof of concept on gel phantoms, the second involved
numeric modeling, and the third involved a pilot trial on
2 subjects. Imaging was performed with a 1.5T MRI machine.
Shear waves were produced with a custom-developed acoustically
driven pneumatic transducer with gradient-echo image
collection gated to the transducer’s motion. Shear wave propagation
were imaged by MRE.
Setting: An MRI research laboratory.
Participants: Two women, one with a 3-year history of
myofascial pain and the other serving as the control.
Interventions: Not applicable.
Conclusions: Our findings suggest that MRE can quantitate
asymmetries in muscle tone that could previously only be
identified subjectively by examination.
Key Words: Magnetic resonance imaging; Myofascial pain;
Rehabilitation.

Problem one: This guy is the Editor for the journal.
Problem two: n=1 in each arm
Problem three: Any validity testing on "Shear waves were produced with a custom-developed acoustically driven pneumatic transducer" Didn't think so.
 
Identification and Quantification of Myofascial Taut Bands
With Magnetic Resonance Elastography

Archives of Physical Medicine and Rehabilitation, Volume 88, Issue 12, December 2007, Pages 1658-1661
Qingshan Chen, MS, Sabine Bensamoun, PhD, Jeffrey R. Basford, MD, PhD, Jeffrey M. Thompson, MD,
Kai-Nan An, PhD
ABSTRACT. Chen Q, Bensamoun S, Basford JR, Thompson
JM, An K-N. Identification and quantification of myofascial
taut bands with magnetic resonance elastography. Arch
Phys Med Rehabil 2007;88:1658-61.
Objective: To explore the feasibility of using a new magnetic
resonance imaging (MRI) technique—magnetic resonance
elastography (MRE)—to identify and quantitate the nature
of myofascial taut bands.
Design: This investigation consisted of 3 steps. The first
involved proof of concept on gel phantoms, the second involved
numeric modeling, and the third involved a pilot trial on
2 subjects. Imaging was performed with a 1.5T MRI machine.
Shear waves were produced with a custom-developed acoustically
driven pneumatic transducer with gradient-echo image
collection gated to the transducer’s motion. Shear wave propagation
were imaged by MRE.
Setting: An MRI research laboratory.
Participants: Two women, one with a 3-year history of
myofascial pain and the other serving as the control.
Interventions: Not applicable.
Main Outcome Measures: MRE images, finite element analysis
calculations, and tissue and phantom stiffness determinations.
Results: Results of the phantom measurements, finite element
calculations, and study patients were all consistent with
the concept that taut bands are detectable and quantifiable with
MRE imaging. The findings in the subjects suggest that the
stiffness of the taut bands (9.00.9KPa) in patients with myofascial
pain may be 50% greater than that of the surrounding
muscle tissue.
Conclusions: Our findings suggest that MRE can quantitate
asymmetries in muscle tone that could previously only be
identified subjectively by examination.
Key Words: Magnetic resonance imaging; Myofascial pain;
Rehabilitation.

This journal publishes some of the finest garbage.
 
I've heard that Racz argues for 18g trigger point injections to "really break up the taught bands".
 
i think you misunderstood Racz --- I am sure he meant to say that you are supposed to use an 18gauge needle followed by catheter adhesiolysis of the fibrosis within the trigger points (and you are to purchase via epimed if possible)...:hardy:
 
It has to be an 18g blunt needle so you don't inject into the sarcoplasmic reticulum.
 
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