What causes the dilation of the LV you see in systolic heart failure? Is the increased EDV causing it to balloon out and the eccentric hypertrophy occurs in repsonse to this? Or does the dilation occur due to hypertrophic mechanisms?
Further, how do remodeling mechanisms in systolic HF become maladaptive?
Further, how do remodeling mechanisms in systolic HF become maladaptive?
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