- Joined
- Aug 31, 2011
- Messages
- 83
- Reaction score
- 4
Cardiovascular stress is one common result of ingesting nicotine by smoking cigarettes. This supports the idea that:
A) nicotine stimulation of the nicotinic acetylcholine receptor is dominant in the sympathetic nervous system
B) nicotine stimulation of the nicotinic acetylcholine receptor is weaker in the sympathetic nervous system
C) nicotine acts directly on cardiac muscle to promote overstimulation and stress
D) nicotine acts indirectly through the central nervous system to produce cardiac stress
I understand the explanation insofar as eliminating C and D, but how can you tell if nicotine stimulation is stronger in the sympathetic NS vs. parasympathic NS just by knowing it creates stress on the heart? Is there something I'm missing?
A) nicotine stimulation of the nicotinic acetylcholine receptor is dominant in the sympathetic nervous system
B) nicotine stimulation of the nicotinic acetylcholine receptor is weaker in the sympathetic nervous system
C) nicotine acts directly on cardiac muscle to promote overstimulation and stress
D) nicotine acts indirectly through the central nervous system to produce cardiac stress
I understand the explanation insofar as eliminating C and D, but how can you tell if nicotine stimulation is stronger in the sympathetic NS vs. parasympathic NS just by knowing it creates stress on the heart? Is there something I'm missing?