The Miseducation of America’s Nurse Practitioners

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He did not have PEA arrest, he had a GCS of 3.


Got it. Was confused by “patient coded” and “ROSC achieved immediately”.

How long was patient in PACU? If vital signs are otherwise stable, sometimes you just need to be patient. That patient would likely have been fine without any narcan. Don’t have to give narcan to every mild overdose.
 
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Couple thoughts and questions for that case.

1. 2mg of dilaudid would be a lot for hip fracture on an opioid naive patient. In a opioid abuser it’s anybody’s guess. Did he receive any additional opioids during the case?

2. What was the respiratory rate and pCO2?

Trying to determine severity of overdose.

Could probably do it with fascia iliaca and 50 of fentanyl
I've seen people be comfortable with block only
I don't think these hurt that much, especially compared to a total hip
 
Got it. Was confused by “ROSC achieved immediately”.

How long was patient in PACU?
If vital signs are otherwise stable, sometimes you just need to be patient. That patient would likely have been fine without any narcan.
I see the confusion. I was talking about a similar case that happened (again with anesthesia) last month. Patient coded that time after she received 0.4 mg of narcan while everyone was waiting for her to wake up; cardiologist showed up and yelled at everyone and gave 2 mg once with almost immediate response.

I don't know how long patient was in the PACU. Timeline showed that ortho dictated his note at 5pm and then called me an hour later.
 
I see the confusion. I was talking about a similar case that happened (again with anesthesia) last month. Patient coded that time after she received 0.4 mg of narcan while everyone was waiting for her to wake up; cardiologist showed up and yelled at everyone and gave 2 mg once with almost immediate response.

I don't know how long patient was in the PACU. Timeline showed that ortho dictated his note at 5pm and then called me an hour later.


Yeah I was referring to the cardiologist’s case. What type of code did they have?
 
Yeah I was referring to the cardiologist’s case. What type of code did they have?
What do you mean? If you are referring that the cardiologist case; I was told patient had a PEA.

Our code blue: Nearest doc has to start the code. ICU attending and on-call hospitalist has go to the code.

When there is a code blue, that cardiologist always shows up even if it's not his patient. And once he shows up, everyone forget about the person who was running the code and wait for him to start giving instructions.

He is a strange dude with probable God complex. I remember I worked night one time and there was a code blue around 2 am. He was already there running the code. The nocturnists also say that happen to them many times as well.

Gotta love medicine in small city/town.
 
What do you mean? If you are referring that the cardiologist case; I was told patient had a PEA.

Our code blue: Nearest doc has to start the code. ICU attending and on-call hospitalist has go to the code.

When there is a code blue, that cardiologist always shows up even if it's not his patient. And once he shows up, everyone forget about the person who was running the code and wait for him to start giving instructions.

He is a strange dude with probable God complex. I remember I worked night one time and there was a code blue around 2 am. He was already there running the code. The nocturnists also say that happen to them many times as well.

Gotta love medicine in small city/town.


Was wondering if it was pure respiratory, respiratory+PEA, or some other rhythm.
 
I find the idea that 0.4mg wasn't enough to be quite remarkable in the above setting. After only 2mg hydromorphone? Wow.

I've never given more than maybe 0.12mg of naloxone to a patient post-op. 40-80mcg increments usually.
 
I see. The pressor probably made the withdrawal looked worse.

I went to a crazy residency where we pushed high dose of narcan all the time.

I was afraid they were playing around with someone that could go into arrest at any minute. I figured out the surgeon called me because he thought they were taking too long. They called me at 6pm and the surgeon dictated his note at 5pm.

It was also amazing that the surgeon did not know anything about the patient; could not even tell me his name.
Why did I think you were an anesthesiologist??
Anyway, most reasonable anesthesiologists would have continued working in more narcan slowly while continuing to examine the patient because I think that’s how most of us were taught since we also have to manage the pain and don’t want them coming off the bed vomiting. Like they do in the movies when they get a needle to the heart!! Hahaha
I certainly wouldn’t have pushed in 1mg at a time, maybe another 0.1 at a time.
But when you say that they were working on him, what were they doing exactly??
 
I see what you are saying; we had a almost similar case (anesthesia again) last month in which the patient coded after she was given only 0.4 while everyone was waiting for her to wake up. Cardiologist showed up yelled at everyone and gave 2mg once and ROSC achieved immediately (though BP shoot up). I have seen that twice and did not want to take that chance.

Indeed that guy SBP shoot up to ~200, but he was also also on levophed that I just started. I stopped the levo gave 10 mg labetalol and his BP gradually went down.
I am sorry. You sound like you work with a bunch of incompetent anesthesiologists. This is weird as hell and bread and butter. Are there always anesthesiologists around? Do people not know how to assess for Narcotic OD and treat??
 
If patients are needing that much narcan, something is very wrong with your anesthesia department.
And they don’t know how to fix their screw ups. They just sit there and watch while not being aggressive enough.
Are these a bunch of new young HCA grads??? What in the damn world???
 
So that patient had PEA arrest with a RR 10-12 and adequate oxygen saturation? To me the picture doesn’t add up. Could there have been another cause of PEA arrest?
I must be stupid here but how does one end up in PEA arrest while still breathing? And Maintaining sats?? This makes no sense here. This isn’t PEA arrest.

NVM- I see the update.
 
I find the idea that 0.4mg wasn't enough to be quite remarkable in the above setting. After only 2mg hydromorphone? Wow.

I've never given more than maybe 0.12mg of naloxone to a patient post-op. 40-80mcg increments usually.
ESRD, Narcotic use history.
But also the anesthesia department sounds incompetent if you ask me.
 
Why did I think you were an anesthesiologist??
Anyway, most reasonable anesthesiologists would have continued working in more narcan slowly while continuing to examine the patient because I think that’s how most of us were taught since we also have to manage the pain and don’t want them coming off the bed vomiting. Like they do in the movies when they get a needle to the heart!! Hahaha
I certainly wouldn’t have pushed in 1mg at a time, maybe another 0.1 at a time.
But when you say that they were working on him, what were they doing exactly??
When I got there I saw they were trying to place an a-line and they told me they already pushed narcan and some phenylephrine (mind you SPB was still in the 60s).

Not sure what transpired from the time surgeon dictated his note to the time he called me by saying 'I just operated on a patient and he has not been able to wake up'
 
ESRD, Narcotic use history.
But also the anesthesia department sounds incompetent if you ask me.
I went back to read their notes. Even before they gave him anesthesia, he was already obtruded from what I can see because they had to call his wife to get consent. He did present to the ED AOx3 from the ED doc note.

Again, I dont know what type of anesthesia was given cause these notes don't appear in EPIC
 
I went back to read their notes. Even before they gave him anesthesia, he was already obtruded from what I can see because they had to call his wife to get consent. He did present to the ED AOx3 from the ED doc note.

Again, I dont know what type of anesthesia was given cause these notes don't appear in EPIC
Yup. They are incompetent. Are they a bunch of new grads? Did they train at HCA? These are actual physicians here we are talking about no? Has to be if you are talking of CAAs involvement.
 
When I got there I saw they were trying to place an a-line and they told me they already pushed narcan and some phenylephrine (mind you SPB was still in the 60s).

Not sure what transpired from the time surgeon dictated his note to the time he called me by saying 'I just operated on a patient and he has not been able to wake up'
Aha. So trying to place an art line in a BP that’s barely palpable…. I am sure that’s easy. Was there any fluid being given?? If they are pushing Phenyephrine why is the BP still low?? Dude, you work with idiots.
What did the pupils look like? We already know the RR was low. Scary place. How big is this hospital???
 
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Aha. So trying to place an art line in a BP that’s barely palpable…. I am sure that’s easy. Was there any fluids being given?? If they are pushing Phenyephrine why is the BP still low?? Dude, you work with idiots.
What did the pupils look like? We already know the RR was low. Scary place. How big is this hospital???
He received ~800 cc by the time I got there. I did not even look at the pupil because reading the last discharge summary about 2-3 wks ago clearly indicated I was dealing with a drug overdose.

That guy was filling up narcotic prescriptions in 2 neighboring states. They told me they already pushed phenylephrine but did not know how much they pushed. I never pushed that drug. I usually start it as a drip.

Hospital of ~250 beds level III trauma center.
 
After reading your responses, maybe I was a little too aggressive. Where I trained, I have seen people pushed much more..
 
It’s basically on the job training that these new arnp get paid for.

Your mileage will vary how competent of a mid level arnp you will get. There are gaps in their knowledgeable many of us physicians can spot right away. But the general public can’t spot.

Don’t get me wrong. There are stuff even I don’t know. I’d tell a patient. Let me go look it up or ask another doc.

I think arrogance will get these mid levels (some of them).

They have to know their limitations and workin the scope of that practice. But sadly many are thrown to the wolves by corporate medicine.
 
The whole point of a slow titration is to avoid a sympathetic explosion resulting in complications such as myocardial ischemia, stroke or SCAPE. 1mg is a bit ambitious and narcan is easy to work in over 10 minutes. No point in bolusing that much unless the patient is comatose on the sidewalk in San Francisco.
Which is essentially what happened.
 
Which is essentially what happened.
Well…. The BP was still in the toilet and the patient was not waking up. GCS of three. They clearly were not moving fast enough in order for the IM/FM doc to come save the day. I would be embarrassed.
So not exactly. Titrate to effect and the effect was not achieved.
 
Did you have to keep redosing the Narcan ? Since the suspected overdose was from dilaudid and it has a longer half life
 
Did you have to keep redosing the Narcan ? Since the suspected overdose was from dilaudid and it has a longer half life
I left the hospital around 1 hour later after given the 1-time dose; did not have to repeat it. I talked to the hospitalist who was assigned to the patient the next day and he told me patient was doing ok and will likely get discharged the following day.
 
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This discussion reminds me of a case where I was on sicu and the medicine team wanted to admit a patient to me for a narcan drip because the patient had surgery 4 days ago. I was like uh why sicu and how do you know it's from opiates? Did you try giving narcan?

They were like no we didn't give narcan because we are afraid of her going back under the effects of opioids but we think she needs a continual drip. I told them that made no sense and then got a phone call later about blocking the admission. I told them that the patient should have went to medical icu and that since the patient was suffering from sepsis unrelated to the operation, I'm pretty sure that the narcan drip wouldn't have helped
 
This discussion reminds me of a case where I was on sicu and the medicine team wanted to admit a patient to me for a narcan drip because the patient had surgery 4 days ago. I was like uh why sicu and how do you know it's from opiates? Did you try giving narcan?

They were like no we didn't give narcan because we are afraid of her going back under the effects of opioids but we think she needs a continual drip. I told them that made no sense and then got a phone call later about blocking the admission. I told them that the patient should have went to medical icu and that since the patient was suffering from sepsis unrelated to the operation, I'm pretty sure that the narcan drip wouldn't have helped
I don't recall seeing any papers showing that naloxone doesn't work for sepsis (this is sarcasm).

From the ED, I don't like admitting people on Narcan drips because I'm worried someone upstairs will forget it's running (i.e. inattentive or overworked nurse) and let it run out when the patient still needs it. I only do intermittent bolus and let the hospitalist order an infusion if they want it.
 
The whole point of a slow titration is to avoid a sympathetic explosion resulting in complications such as myocardial ischemia, stroke or SCAPE. 1mg is a bit ambitious and narcan is easy to work in over 10 minutes. No point in bolusing that much unless the patient is comatose on the sidewalk in San Francisco.
SCAPE?
 
Yup. They are incompetent. Are they a bunch of new grads? Did they train at HCA? These are actual physicians here we are talking about no? Has to be if you are talking of CAAs involvement.
HCA?
 
Never heard of this either.
I see it with some regulatory in the unit from hypertensive emergencies. The ED or hospitalist will see the flash edema, think HF exacerbation, and try to aggressively diurese and either ignore the BP, or try to throw a ton of labetalol at that, as well. Unfortunately, diuresis can be counterproductive or ineffectual, as once you lower the afterload, improving forward flow, they can then become hypotensive from any volume loss. Instead, staring nicardipine and bridging with BIPAP for a few hours fixes everything.

Meth-induced cardiomyopathy plus acute opioid withdrawal from high dose narcan is a perfect setup for this where I practice.
 
I see it with some regulatory in the unit from hypertensive emergencies. The ED or hospitalist will see the flash edema, think HF exacerbation, and try to aggressively diurese and either ignore the BP, or try to throw a ton of labetalol at that, as well. Unfortunately, diuresis can be counterproductive or ineffectual, as once you lower the afterload, improving forward flow, they can then become hypotensive from any volume loss. Instead, staring nicardipine and bridging with BIPAP for a few hours fixes everything.

Meth-induced cardiomyopathy plus acute opioid withdrawal from high dose narcan is a perfect setup for this where I practice.
Why the damn fancy name? This is simply Hypertensive Induced Pulmonary edema. Or Edema from severe Hypertension.

Why do people trying to make things complicated? To make themselves feel brilliant??
I thought it was something new.
Sympathetic crashing? What’s the crash? The edema?
SMH
 
Hospital Corporation of America.
Are you American?? Although I met people in the Midwest on my locum journey who have never heard of this as well.
Deep South. Do they churn out crappy residents?
 
Why the damn fancy name? This is simply Hypertensive Induced Pulmonary edema. Or Edema from severe Hypertension.

Why do people trying to make things complicated? To make themselves feel brilliant??
I thought it was something new.
Sympathetic crashing? What’s the crash? The edema?
SMH
You answered your own question. Academics make up new terms for uncommon conditions every few years to feel important that they 'clarified' the condition by giving it a new name.

The crashing is because it tends to be a very acute presentation, and they can go from looking fine to profoundly dyspneic with sats in the 70s within a minute or two of the sympathetic surge that lead to the greatly increased afterload.
 
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