- Joined
- May 20, 2016
- Messages
- 57
- Reaction score
- 88
I'm having a hard time conceptually making sense of high E bonds and energy release.
I've learned that bonds store energy and so was under the impression that breaking a bond would release Energy but it's the opposite.
In ATP hydrolysis, it's favorable and exergonic. I've read another thread explaining that E released by ATP hydrolysis is actually due to a small amt of E being put into breaking the weak bonds of ATP followed by the release of a large amt of E due to formation of stable, low E bonds, thus resulting in a net energy release making the rxn favorable.
Why are the ATP phosphanhydridic bonds considered weak but high E?
Also, if this is true, are all metabolic rxns that use ATP to push a rxn, getting the E from not the breaking of the bonds but the formation of a bond with part of the ATP?
I've learned that bonds store energy and so was under the impression that breaking a bond would release Energy but it's the opposite.
In ATP hydrolysis, it's favorable and exergonic. I've read another thread explaining that E released by ATP hydrolysis is actually due to a small amt of E being put into breaking the weak bonds of ATP followed by the release of a large amt of E due to formation of stable, low E bonds, thus resulting in a net energy release making the rxn favorable.
Why are the ATP phosphanhydridic bonds considered weak but high E?
Also, if this is true, are all metabolic rxns that use ATP to push a rxn, getting the E from not the breaking of the bonds but the formation of a bond with part of the ATP?