- Joined
- Jun 3, 2007
- Messages
- 4,694
- Reaction score
- 3,096
I heard that they did defibrillate.I would think so, but it’s interesting that the rhythm wouldn’t be disrupted/degenerate upon impact.
He got up and seemed okay for a second, and then you can see when he starts to feel weak and collapse. To me, it seems more in line with vasovagal syncope after getting up too quickly and some sort of bigger issue after that. They did do CPR on the field so that makes me think maybe it wasn’t a rhythm issue (if they didn’t have to defibrillate), but who knows?
I thought they did a chest compressions and shock therapy.I heard that they did defibrillate.
I agree. I’ve got a partner that tells a story of pacing a pt in complete heart block with precordial thumps. More or less this would be a pacer spike on a T-wave. If you look a the hit it wasn’t particularly violent but at the perfect angle for a precordial thump.seems most likely
Tackle didn’t look too bad, also wearing pads. My money is on some kind of arrhythmia. HCM? If so the prognosis is fairly poor. CPR for 9 minutes on a healthy guy without ROSC? Likely a shockable rhythm. If the time to defibrillation was fairly long, no bueno. Hopefully good compressions. Poor guy.
I can't imagine a better prognosis than a witnessed cardiac arrest in an otherwise healthy 24 year old. Apparently they got him onto a stretcher before having to start CPR so the only question is how long was he without CPR and when did they recognize he had no pulse.
If anything the fact that his vitals are stable and got ROSC rule out neck injury, transection, rupture or anything immediately fatal. My bigger concern is the fact that 12 hours later there hasn't been word about his neuro status which would likely indicate some sort of delayed neurological recovery. Probably cooling him now.
The thing that worries me is that there are reports of him only getting CPR after they got him on the stretcher. It's always possible that there was some confusion about what was going on, but you'd have expected CPR to be the first thing administered in that kind of situation.Yeah I'm surprised they haven't announced he's woken up given how soon compressions started. One thing he has going against him as far as organ protection though is that his body temperature and metabolic rate were probably an order of magnitude higher than the average schlub who collapses while doing nothing. But again, his ischemic time must've been short given how soon they got the AED.
I don't think him standing up rules out arrhythmia. When we to TAVRs and they start rapid pacing, I occasionally see the MAP drop to the 30s momentarily and the patient will keep talking to me, or be looking around. Sometimes they wig out for a moment (start moving head around) as the pacing stops and pressures have been low for 20 seconds.I would think so, but it’s interesting that the rhythm wouldn’t be disrupted/degenerate upon impact.
He got up and seemed okay for a second, and then you can see when he starts to feel weak and collapse. To me, it seems more in line with vasovagal syncope after getting up too quickly and some sort of bigger issue after that. They did do CPR on the field so that makes me think maybe it wasn’t a rhythm issue (if they didn’t have to defibrillate), but who knows?
Lying on a table w/ a MAP of 30 =/= standing w/ MAP of 30I don't think him standing up rules out arrhythmia. When we to TAVRs and they start rapid pacing, I occasionally see the MAP drop to the 30s momentarily and the patient will keep talking to me, or be looking around. Sometimes they wig out for a moment (start moving head around) as the pacing stops and pressures have been low for 20 seconds.
My point is, from the hit to the time he fell over was literally only a few seconds. It would not be unreasonable to assume his brain and body could have been working on a tiny bit of oxygen reserve. Additionally it could have been a progressing arrhythmia that was perfusing for a moment but degraded to an unstable arrhythmia pretty quickly.Lying on a table w/ a MAP of 30 =/= standing w/ MAP of 30
Typically training is to cut off pads and flip them.commotio cordis seems likely. hopefully they recognized it quickly, got him on a backboard gave good quality cpr. I doubt it's easy giving good cpr with shoulder pads on (which come across the chest) plus whatever other equipment professional football players are wearing.
Agreed. Standard of care going forward should be Pre-game locker room A lines for everyone.My point is, from the hit to the time he fell over was literally only a few seconds. It would not be unreasonable to assume his brain and body could have been working on a tiny bit of oxygen reserve. Additionally it could have been a progressing arrhythmia that was perfusing for a moment but degraded to an unstable arrhythmia pretty quickly.
I saw the hit and my initial thought was vasovagal as well.
From the way the hit was it doesn't look like commotio cordis, like if you think about how the heart sits inside the chest and how he was bracing for the impact. Higgins made contact on the left shoulder and neck area and Hamlin landed on his left arm/side. None of that screams direct blunt contact to the heart to me.
More likely it's vasovagal -> apnea -> hypoxia rather than arrhythmia -> hypotension -> cardiac arrest.
But it's kinda impossible to tell. I hope he turns out ok.
I saw the hit and my initial thought was vasovagal as well.
From the way the hit was it doesn't look like commotio cordis, like if you think about how the heart sits inside the chest and how he was bracing for the impact. Higgins made contact on the left shoulder and neck area and Hamlin landed on his left arm/side. None of that screams direct blunt contact to the heart to me.
More likely it's vasovagal -> apnea -> hypoxia rather than arrhythmia -> hypotension -> cardiac arrest.
But it's kinda impossible to tell. I hope he turns out ok.
Vasovagal hard enough to break all that sympathetic tone? His HR was prob 110-120 at the time of the hit. And even if he did vasovagal > syncopize, why would he have prolonged apnea? Something else would have to be wrong for him to stop breathing. I watched the hit on replay a bunch of times. I don’t think it was when they hit the ground, it was when his opponents helmet first made contact with his chest. That was the “commotio cordis” moment. And as others have said, a relatively minor hit can generate the joules necessary to elicit a “pacer spike”, and in this instance it hit at just the right millisecond to cause an R on T. That’s my hypothesis at least. As you said though, no way to really know.I saw the hit and my initial thought was vasovagal as well.
From the way the hit was it doesn't look like commotio cordis, like if you think about how the heart sits inside the chest and how he was bracing for the impact. Higgins made contact on the left shoulder and neck area and Hamlin landed on his left arm/side. None of that screams direct blunt contact to the heart to me.
More likely it's vasovagal -> apnea -> hypoxia rather than arrhythmia -> hypotension -> cardiac arrest.
But it's kinda impossible to tell. I hope he turns out ok.
eh, idk... vasovagal... then 9 minutes of CPR?
Helmet to carotid body on a 23 year old.Vasovagal? What exactly was the vagal stimulus. How does that lead to 9 minutes of CPR?
Just listening to an interview with one of the NFL physicians that CPR started immediately and the AED was used before the ambulance arrived on the field.The thing that worries me is that there are reports of him only getting CPR after they got him on the stretcher. It's always possible that there was some confusion about what was going on, but you'd have expected CPR to be the first thing administered in that kind of situation.
Looking at the video, the football was between Hamlin's chest and the WR when he was tackled. It is possible the football may have provided the source for the more focused blunt contact.I saw the hit and my initial thought was vasovagal as well.
From the way the hit was it doesn't look like commotio cordis, like if you think about how the heart sits inside the chest and how he was bracing for the impact. Higgins made contact on the left shoulder and neck area and Hamlin landed on his left arm/side. None of that screams direct blunt contact to the heart to me.
Upon looking further, I agree with the above and think it is more likely that commotio occured during the initial impact.
It is more noticeable from the sideline camera view.
Looking at the video, the football was between Hamlin's chest and the WR when he was tackled. It is possible the football may have provided the source for the more focused blunt contact.
Goodness some of the theories put forward in this thread are bizarre. Vasovagal? What exactly was the vagal stimulus. How does that lead to 9 minutes of CPR?
Cessation of cerebral blood flow is not instantaneous, whether sitting or standing.
There are a bunch of videos you can find on YouTube of this phenomenon- dude gets hit in chest, staggers around for a bit, then collapses a few seconds later
I agree that his particular metabolic state would probably cause him to be somewhat hypoxic.I wonder how much oxygen he using at that particular time that could have led to faster anoxic brain injury? Just a theory
He has the blessing of youth.I agree that his particular metabolic state would probably cause him to be somewhat hypoxic.
But I would imagine effective CPR would give him ample oxygen supply given his lungs are probably top notch.
The time between circ arrest and start of quality CPR is probably gonna determine how well he does long term.
No, anybody whose ventilated post arrest. Besides which hypothermia is rarely practiced anymore- mostly TTM (35-36 degrees).haven't been in the unit in years now but what are the requirements for hypothermia? any minimum exam findings or anything that excludes you from being cooled?
No, anybody whose ventilated post arrest. Besides which hypothermia is rarely practiced anymore- mostly TTM (35-36 degrees).
I don't do TTM for everybody that's vented. I just shut off sedation immediately and if I get a neuro status avoid fevers. Data is way too murky.No, anybody whose ventilated post arrest. Besides which hypothermia is rarely practiced anymore- mostly TTM (35-36 degrees).
or just he never never needed the 100% fio2 or maybe he did from the cpr itselfI don't do TTM for everybody that's vented. I just shut off sedation immediately and if I get a neuro status avoid fevers. Data is way too murky.
Reports out there are showing he's down to 50% FiO2 from 100% so I'm guessing he may have had some contusion and pulmonary edema contributing
I don't do TTM for everybody that's vented. I just shut off sedation immediately and if I get a neuro status avoid fevers. Data is way too murky.
Reports out there are showing he's down to 50% FiO2 from 100% so I'm guessing he may have had some contusion and pulmonary edema contributing
Very large trial (called TTM2) came out recently which has settled the question for many ICUs. Essentially the data showed that maintenance of normothermia (avoidance of fever) has similar outcomes to therapeutic hypothermia but with fewer side effects (arrhythmia, hypotension). It's also cheaper and less labor intensive.haven't been in the unit in years now but what are the requirements for hypothermia? any minimum exam findings or anything that excludes you from being cooled?