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My dad has had an anterio-septal (V1-V3) heart attack 7 months ago. He was a heavy smoker, not active at all and also his blood cholesterol level was increased.
He's being monitored every 2-3 months by his cardiologist. He had an EF of 30% at the time of the heart attack.His last echo showed an increase of EF up to 45%, otherwise he's clinically doing just fine, no dyspnea on exertion or any kind of pain at all, no orthopneae, no peripheral edema, nothing that relates to HF.
He definitely stopped smoking now and walks briskly 5-8 miles every day. He's taking Clopidogrel, Aspirin, Atorvastatin, Metoprolol and Metformin momentally.
I am just concerned about his prognosis. I know that compensatory mechanisms underwent their function to maintain a desirable CO, but I am a medical student and based on what I've read and studied, eventually these compensatory mechanisms will weaken heart muscle and other tissues,thus HF is inevitable. Is this true? Please clarify and tell me the truth even though it may be sad.
Thanks in advance,
He's being monitored every 2-3 months by his cardiologist. He had an EF of 30% at the time of the heart attack.His last echo showed an increase of EF up to 45%, otherwise he's clinically doing just fine, no dyspnea on exertion or any kind of pain at all, no orthopneae, no peripheral edema, nothing that relates to HF.
He definitely stopped smoking now and walks briskly 5-8 miles every day. He's taking Clopidogrel, Aspirin, Atorvastatin, Metoprolol and Metformin momentally.
I am just concerned about his prognosis. I know that compensatory mechanisms underwent their function to maintain a desirable CO, but I am a medical student and based on what I've read and studied, eventually these compensatory mechanisms will weaken heart muscle and other tissues,thus HF is inevitable. Is this true? Please clarify and tell me the truth even though it may be sad.
Thanks in advance,
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